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Biomedical subjects

A Azzam

Publications and source records attributed to A Azzam.

5 recordsLinked to original sources
PubMed1991

Interaction of naturally occurring aflatoxins in poultry feed and immunization against fowl cholera.

A total of 1,175 poultry feed samples originating from different farms were analyzed for aflatoxin. Poor growth rate and reduced egg production were the main complaints. The rate of contamination with aflatoxin ranged from 10 to 54% of all samples. Of samples examined 30.7% proved positive for aflatoxin with a concentration ranging from 1 to 2,000 ppb. Outbreaks of fowl cholera were diagnosed on two farms where aflatoxin was detected in the rations used. The impact of aflatoxin in the feed on the efficacy of immunization against fowl cholera was monitored by a hemagglutination test and the titers of the involved farms were compared with experimental groups fed on aflatoxin-free rations and vaccinated with the same polyvalent fowl cholera bacterin. The antibody titers of the experimental groups were 4 to 15 times higher than those of the involved farms.

Aflatoxin B1↗
PubMed1988

[Thyroid ectopia and Basedow's disease].

Graves' disease was diagnosed in a 35-year old woman with a history of myxoedema in childhood. Clinical data, ultrasonography and radioisotope scanning showed that the disease had developed in an ectopic subhyoid thyroid gland. Although numerous thyroid diseases associated with an ectopic thyroid have been described, Graves' disease does not seem to be frequent since only one case has been published so far.

Adult↗
PubMed1987

[Homozygous infant in a family with hereditary protein C deficiency].

We report on a female infant homozygous for protein C deficiency in a Jordanian family with frequent intermarriage. A protein C antigen of 0.6% was determined. The parents first noticed painful nodular indurations in subcutanous tissue as well as blue-red skin coloration at the age of 6 months. The girl repeatedly suffered from microthrombotic events in parts of the body with large areas of subcutaneous fat. In contrast, the numerous heterozygous carriers with partial protein C deficiency did not show an increased tendency to thrombosis. From the history an autosomal-recessive inheritance may be inferred. Other authors reporting on homozygous cases also postulate the presence of a recessive gene. It is of interest that the infant described here differs from those in other case reports in the age at manifestation of the disease. The homozygous infant showed the first symptoms as late as the age of 6 months, whereas other case reports describe severe symptoms immediately after birth. All symptoms of disease were treated successfully with prothrombin complex concentrate without additional heparin protection. Microthrombotic events subsided quickly, and a large ulcer in the left flank healed almost completely within 6 days.

Adult↗
PubMedDate not supplied

[Extra-arachnoid subdural injection, an accident of peridural anesthesia].

Two cases are presented, with accidents of peridural anesthesia, Anesthesia of the entire brain stem, paralysis of the intercostal muscles, and of the upper limbs, apnoea and miosis that developed later indicate an extension of the anesthetic effects far higher that it could have been expected considering the technical details of the procedure. On the other hand the absence of any durable coma, of extreme mydriasis, and of severe arterial hypotension, as well as the relatively rapid retrocession of the additional effects of the administration of an anesthetic exclude the possibility of total rachianesthesia, and it can be concluded that there was an accidental injection of anesthetic in the extra-arachnoid subdural space.

Adult↗