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A B Gekht

Publications and source records attributed to A B Gekht.

41 records · Page 3Linked to original sources
PubMedDate not supplied

Clinical-neurophysiological features of motor lesions in patients with post-stroke epilepsy.

Twenty-two patients with post-stroke epilepsy (group 1) were studied, along with 30 stroke patients without epilepsy (group 2). Bilateral (on both the paralyzed and intact sides) decreases in the central conduction time (CCT) along the pyramidal tract were found in group 1, which were not seen in group 2, who had similarly severe motor lesions (p < 0.01). The tendency to decreased CCT was also seen 5-6 days after ischemic stroke in patients without epileptic manifestations, though CCT in this group increased by day 10-14; low values persisted in group 1 for prolonged periods. The facilitation differentiation (deltaF, the difference in CCT between resting and effort conditions) was also found to increase. In patients with post-stroke epilepsy, motor lesions were characterized by higher levels of muscle tone than in other stroke patients, though the levels of paralysis were similar.

Aged↗
PubMedDate not supplied

[The dependence of the cortical topography of visual evoked potentials on the stimulation frequency].

Cortical topography of steady-state flash visual evoked potentials (fVEP) was compared with topography of transient fVEP and pattern VEP (pVEP) in 17 normal subjects. It was found that at low (up to 20 Hz) frequencies fVEP demonstrate extensive dissemination on the scalp. At higher frequencies (30 Hz or higher) fVEP were registered mostly in the occipital area similarly as transient pVEP. These results suggest that the neural structures responsible for the generation of transient pVEP and steady-state fVEP of 30 Hz and higher frequencies are anatomically identical.

Adolescent↗
PubMedDate not supplied

[Tibial neuromodulation in the treatment of neurogenic detrusor hyperactivity in patients with Parkinson's disease].

Twenty nine patients with neurogenic detrusor overactivity due to Parkinson's disease have undergone percutaneous tibial neuromodulation. The treatment consisted of percutaneous insertion of a 34-gauge solid stainless steel needle at a point of 5 cm cephald from the medial malleolus. The patients were treated weekly for 30 minutes. A subjective effect was assessed using a dairy and I-PSS score. Objective results were evaluated by cystometry. We observed a decrease in the average voiding frequency, number of leakage episodes after 12 sessions and 6 months of the tibial neuromodulation. An over 50% symptomatic improvement was achieved in 26 of 29 patients including 6 patients who were refractory to anticholinergic agents and 9 men with benign prostatic hyperplasia. Tibial neuromodulation is an effective treatment in patients with neurogenic detrusor overactivity due to Parkinson's disease.

Aged↗