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Biomedical subjects

A Briese

Publications and source records attributed to A Briese.

5 recordsLinked to original sources
PubMed1996

[Mobile slaughter facilities].

Mobile slaughter is commonly discussed as alternative method to slaughter avoiding transportation stress in slaughter animals. In 1994 mobile slaughter became part of the coalition-contract between the two major parties, SPD and GRUNE, in the Department HESSEN. In the article the actual principles, problems to cope with EU-legislation and hygiene-standards, chances and risks of mobile slaughter are discussed. Assuming that some of the major problems concerning hygiene, workers security and waste-deposit can be solved, mobile slaughter seems to bring an improvement in animal welfare and advantages in related meet quality. The slightly higher costs may be acceptable in welfare-interested, high-quality market sections.

Abattoirs↗
PubMed1995

[Fatal accident to a horse from a narrow transverse beam in the box door].

It is reported on the fatal accident of a horse due to a cross-beam too low above the door of the stable, in which the animal suffered several skull fractures and a tear of the musculus longus capitis from the base of the skull. The aim of this article is, to supply a contribution to the establishment of minimal requirements for the housing of horses, in this case for the request of the minimum height of stable doors and ceilings as well as for constructions on horse transporters. On the grounds of this example, the natural behaviour of horses is discussed at the hand of the literature and the efforts are shown to improve the existing inadequacies in the housing and transport of horses with regard to the judicial situation. In the light of section 2 of the animal protection law, as to the construction of horse stables the importance is especially emphasized to consider the panic behaviour in connection with abrupt flight reactions as physiologic in the prevention of accidents.

Accidents↗
PubMed1994

[Animal welfare in the slaughterhouse].

The importance of correct handling of slaughter animals at the farm, on transport and in the slaughterhouse is described. The status of animal welfare in slaughterhouses, the relations between animal welfare, preslaughter animal condition and meat quality are discussed. Possibilities of better information exchange by integrated quality systems are shown. A summary of the first-year-work experiences of the Beratungs- und Schulungsinstitut für tierschonenden Umgang mit Nutz- und Schlachttieren (bsi) gives an overview of handling and related animal welfare problems during the slaughter process.

Abattoirs↗
PubMedDate not supplied

Inhibition by aspirin of the stimulated PGI2 synthesis after administration of kallikrein to hypertensive patients (new aspects of chemical pathogeny concerning the messenger systems: A and C).

The vascular smooth muscle cells belong to the category of "bidirectionally controlled cells", which can be activated (leading to contraction) by some first-messengers (such as Catecholamine-alpha 1, or Angiotensin II) in connection with a recently discovered intracellular second-messenger system-C (sms-C), namely, Diacylglycerol-dependent Protein Kinase-C + Calcium mobilization. On the other hand, these cells can be inhibited (leading to relaxation) by other first-messengers (such as Catecholamine-beta or PGI2) related to the classical "second-messenger system-A" (sms-A), namely: Ns--Adenylate Cyclase--cAMP--Protein Kinase-A. It is also known that kallikrein (via kinins) can stimulate PGI2 synthesis, and implicitly sms-A, relaxing the vascular tone by counteracting the opposite pressor effect of sms-C. In the four h samples of urine, collected before and after administration of kallikrein (i.m., 40 IU) to six hypertensive patients, we measured (RIA): 6-keto-PGF1 alpha, the stable metabolite of PGI2. The results 5.86 +/- 1.25 vs 9.94 +/- 0.6 pg/mL +/- SEM (p less than 0.05) indicate a rise in PGI2 synthesis. However, no such effect was obtained when the same patients were given aspirin (4 g in divided doses) one day before repeating the above test: 5.59 +/- 1.97 vs 6.85 +/- 1.28 (NS). We may conclude that administration of kallikrein can stimulate PGI2 synthesis, which, in turn, can be blocked by relatively high (= nocive) doses of aspirin. Some new aspects of chemical physiology, pathogeny and systematization of the two major messenger systems, MS-A and MS-C, are discussed.

6-Ketoprostaglandin F1 alpha↗