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Biomedical subjects

A Di Lorenzo

Publications and source records attributed to A Di Lorenzo.

9 recordsLinked to original sources
PubMed2001

Integrable model for interacting electrons in metallic grains.

We find an integrable generalization of the BCS model with nonuniform Coulomb and pairing interaction. The Hamiltonian is integrable by construction since it is a functional of commuting operators; these operators, which therefore are constants of motion of the model, contain the anisotropic Gaudin Hamiltonians. The exact solution is obtained diagonalizing them by means of Bethe ansatz. Uniform pairing and Coulomb interaction are obtained as the "isotropic limit" of the Gaudin Hamiltonians. We discuss possible applications of this model to a single grain and to a system of few interacting grains.

Journal Article↗
PubMed1988

Trends in combustion technology in relation to health risk.

The general aspects of air pollutant formation during combustion are summarized, and emphasis placed on smoke emissions highly suspected of inducing cancer in human beings. A health risk index is defined, taking into account both quantities and toxicities of emission compounds. Pollutant formation during real combustion processes is discussed considering different fuels and different combustion devices both for stationary applications and for transportation. The possible technological actions for emission reduction with regard mainly to soot and polynuclear aromatic hydrocarbons are described for different cases. The paper ends with some considerations of the prospects for research in the field of combustion and on the strategies of cooperation between combustion scientists and toxicologists.

Benzene↗
PubMedDate not supplied

Elevated alanine aminotransferase in blood donors: role of different factors and multiple viral infections.

Many different aetiological agents stimulate alanine aminotransferase (ALT) production. Viral markers and other aetiologies were investigated in 2166 individuals, randomly selected from 10,000 consecutive blood donors. Elevation of ALT was found in 10.8% of subjects. Grouping donors according to ALT level and correlating with, respectively, hepatitis B core antibody (HBcAb), cytomegalovirus antibody alone, or associated with HBcAb, showed similar findings (high ALT 11.1%, normal 11.6%; high 85.4%, normal 81.4%; high 10.2%, normal 11.0%, respectively). Hepatitis C virus (HCV) antibody was found to be significantly associated with elevated ALT levels (high 1.7%, normal 0.26%). Other causes of ALT elevation were alcohol abuse (17%), obesity (25%) and dyslipidaemia (38%), but in 11% there was no obvious aetiology. Although HCV is a rare cause of elevated ALT in blood donors, it seems to be the only virus, among those tested, to account for liver damage. This may be due to the non-protective role of HCV antibody, the low specificity of ALT, or the pathogenic role of uninvestigated viruses.

Alanine Transaminase↗