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Biomedical subjects

A F Reynolds

Publications and source records attributed to A F Reynolds.

At least 37 records · Page 2Linked to original sources
PubMed1980

Lower cranial nerve palsies due to Pseudomonas osteomyelitis of the skull base.

A patient with pseudomonas osteomyelitis of the base of the left posterior fossa is reported. His clinical course was one of progressive paresis of the left 8th, 10th, and 11th cranial nerves. There have been three prior reports of osteomyelitis of the base of the skull not in contiguity with an infected paranasal sinus. Our patient subsequently developed osteomyelitis in the 3rd to 5th cervical vertebrae.

Accessory Nerve↗
PubMed1980

Generous craniectomy for Kleeblattschädel anomaly.

In the case presented, Kleeblattschädel, or cloverleaf skull deformity, was treated successfully with a two-staged, generous craniectomy. The pertinent aspects of this rare anomaly are reviewed in light of information from other cases described previously. The present case illustrates that an early surgical approach of subtotal calvariectomy may improve the commonly predicted poor prognosis in patients with Kleeblattschädel anomaly if the clinical severity of the disease is not immediately overwhelming.

Abnormalities, Multiple↗
PubMed1980

Adenocarcinoma cells trapped on a millipore filter in a patient with meningeal carcinomatosis.

A patient with meningeal carcinomatosis and hydrocephalus secondary to metastatic adenocarcinoma of the breast is presented. A ventriculoperitoneal shunt with a 3-microns Millipore filter was placed to prevent abdominal seeding of tumor cells. Within 5 days the shunt became plugged, requiring removal of the plugged filter. Tumor cells and hematogenous cells were found on the ventricular surface of the filter. The histological findings and the electron microscopy of these cells are presented. Millipore filters should probably not be placed in patients with carcinomatous meningitis.

Adenocarcinoma↗
PubMed1980

Postpartum acute subdural hematoma; a probable complication of saddle block analgesia.

The author present a patient who developed an acute intracranial subdural hematoma approximately 35 hours after an uncomplicated delivery under saddle block analgesia. The proposed mechanism is that of cerebrospinal fluid efflux through the lumbar puncture site during uterine contractions and straining; shift of the intracranial structures, especially veins; venous hypertension; venous rupture; and subdural hematoma formation.

Acute Disease↗
PubMed1980

Persistent otic artery.

A patient with a persistent otic artery is presented. The radiographic anatomy is presented in the anteroposterior as well as the lateral plane. The significance of the otic artery is twofold. First, it is important in understanding the embryogenesis of the cerebral vessels. Secondly, it is an anatomical variant that is important in performing and evaluating cerebral arteriograms.

Aged↗
PubMed1978

A complication of nasogastric intubation: intracranial penetration.

A patient with severe maxillofacial trauma had a nasogastric tube inserted into the intracranial cavity. Two such case reports were found in the literature and in both instances the patients expired. The present patient is believed to be the first who survived intracranial passage of a nasogastric tube.

Adult↗
PubMed1978

Aphasia and left thalamic hemorrhage.

Left thalamic hemorrhage as a cause of aphasia has not been widely recognized. Large thalamic hemorrhages cause coma, making speech examination impossible; smaller thalamic hemorrhages were difficult to document until recent diagnostic advances. Nine cases of thalamic hemorrhage with aphasia have been described in the literature. This report presents four additional cases. These patients had acute onset of aphasia, supranuclear paralysis of upward gaze, right hemisensory deficits, and mild right hemiparesis. Three of the four patients responded to ventriculostomy drainage with rapid clearing of the supranuclear paralysis of upward gaze, and two later required placement of permanent ventricular shunts. After 1 year, two patients exhibited no clinically detectable speech malfunction and the other two were severely aphasic. The hemiparesis, hemisensory deficits, and ocular pareses all cleared. These cases are discussed with respect to present models of the role of the thalamus in speech.

Adult↗