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Biomedical subjects

A I Berestov

Publications and source records attributed to A I Berestov.

At least 19 recordsLinked to original sources
PubMed1993

[Neuro-AIDS in children].

The paper analyzes the results of studying nervous diseases in children with HIV infection. A total of 57 children aged 1.5 to 16 years who had different stages of AIDS were examined. HIV-related encephalopathy, encephalopathy with the polyneuropathy syndrome, encephalomyelopolyneuritis, mixed AIDS-related encephalopathy and perinatal encephalopathy, as well as the myopathic syndrome were identified. The affliction of the nervous system was more severe with an earlier history of infection. The clinical syndromes were supported by electromyographic indices.

AIDS Dementia Complex↗
PubMed1989

[Neurosonography of the brain in infectious toxicoses in infants].

Brain neurosonography was performed in 15 babies with toxicosis-+-linked nervous disorders. Three types of the brain structure were established in the patients: 1, marked brain swelling in all patients; 2, concomitant perinatally induced brain structures' changes; 3, intracranial hemorrhage focuses resulting from the DIC syndrome. The technique is useful for precise diagnosis and prognosis of the outcome of the neurotoxic coma.

Acute Disease↗
PubMed1988

[Specific brain antigens--an index of the permeability of the hemato-encephalic barrier in comatose states in children].

In 39 children with coma caused by infectious toxicosis, purulent meningoencephalitis and closed head trauma the brain-specific antigens (L1M and L2M globulins) contents were studied in the blood serum and cerebrospinal fluid (CSF). On days 3 to 5 after the head trauma L1M and L2M were found irregularly. In meningoencephalitis L1M and L2M were detected with a lesser delay. In infectious toxicosis L1M and L2M appeared in blood serum and CSF regularly and early. The data suggest that appearance of brain-specific antigens in blood and CSF in coma reflect a rise in the blood-brain barrier permeability to CSF-blood transport.

Alpha-Globulins↗
PubMed1988

[Acute hepatic encephalopathy (Reye's syndrome) in children)].

Subjects of clinical and biochemical investigation were 8 children aged 4.5 months to 12 years suffering from hepatocerebral syndrome which had developed after acute respiratory infection, encephalitis, acute appendicitis. Methods of pathogenic therapy and early diagnosis are offered.

Acute Disease↗
PubMed1987

[Crystallographic study of cerebrospinal fluid in closed craniocerebral injuries in children].

The authors examined 65 children with closed head trauma of varying severity and 68 children with other acute neurological diseases. A new method of the diagnosis and assessment of the severity of closed craniocerebral injury, namely, crystallographic examination of the patients' cerebrospinal fluid, was used in the study. The method helped to establish the clinical forms of closed head trauma and to determine its severity and the presence of complications.

Adolescent↗
PubMed1985

[Cerebral contusions in injuries with fractures of the bones of the skull in children].

The authors analyzed 205 cases of skull fractures in children. Linear, depressed and comminuted skull fractures as well as ruptures of the cranial commissures were revealed. Skull fractures in children were characterized by specific features depending on the age, type of fractures and the mechanism of injury and were always attended with contusions of the brain. To determine the degree of brain contusions, the authors carried out a comprehensive examination with the use of electrophysiological and modern laboratory methods (determination of blood plasma prostaglandins).

Adolescent↗
PubMed1985

[Neurotoxic syndrome in infectious diseases in children].

On the material of 72 children with infectious toxicoses the authors describe the encephalic syndrome as a manifestation of the neurotoxic syndrome. The neurotoxic syndrome was studied clinically and morphologically. A marked syndrome was characterized by a considerable edema of the brain and its stem and not infrequently by the vedging of the cerebellum into the great occipital orifice. Hemispheric focal symptomatology was typically associated with intracranial hemorrhage which was considered as a manifestation of the DIC syndrome. The authors believe that the neurotoxic syndrome in infectious toxicoses should be considered as general cerebral disorders of noninflammatory nature manifested in non-specific morphofunctional and metabolic disturbances.

Brain↗
PubMed1983

[Clinico-biochemical parallels in neurotoxicoses in children].

The authors present the results of examining the plasma lipids in 62 children with an infectious neurotoxic syndrome. The children were suffering from acute respiratory viral infections, lobular pneumonia, or intestinal infections. Hyperlipidemia and dysphospholipidemia were revealed: the former was due primarily to a rise of the content of non-saturated fatty acids and triglycerides, and the latter to a drop of the lecithin and cephaline levels. These changes correlated with the disease gravity. No substantial differences in the plasma lipid spectrum were revealed in the children with the neurotoxicoses due to the acute respiratory infections, pneumonias, and intestinal infections, however, in the latter cases a more marked rise of the triglyceride content was noted.

Child, Preschool↗