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Biomedical subjects

A Iu Ratner

Publications and source records attributed to A Iu Ratner.

At least 19 recordsLinked to original sources
PubMed1990

[Neurologic evaluation of unconditioned reflexes in the newborn].

The authors examined 197 neonates with different lesions of the brain and spinal cord. The conclusion was made that investigation of unconditioned reflexes can make a valuable contribution to the topical diagnosis. In cerebral lesions, search, sucking and lip reflexes disappear or dramatically reduce. In lesions of the cervical part of the spinal cord, these reflexes are preserved. However, grasp, Robinson's, Moro's, defense and palmarmouth reflexes disappear. In lesions of the lumbar enlargement, support, automatic walking, Perez's, Bauer's, jerk, crossed extension, and inferior grasp reflexes are absent, whereas the main reflexes are absolutely preserved. In the authors' opinion, the data obtained open up new potentialities in neurology of the newborn.

Humans↗
PubMed1984

[Secondary changes in the hip joints of children with progressive muscular dystrophy].

The authors state that a marked hypotonia of the lower extremity muscles in children with progressive muscular dystrophy may lead to the development of secondary dislocations and semi-dislocations of the femur, which were detected in 18 of the 32 patients examined. Most of the patients had Werdnig-Hoffmann's spinal amyotrophy which was related to an especially gross muscular hypotonia.

Child↗
PubMed1981

[Clinical picture and diagnosis of a myotonic syndrome in children resulting from birth injury].

The authors observed 68 ill children with pronounced muscular hypotonia (myotonia), who were thoroughly examined. The authors describe a group of cervical syndromes and present the symptomatology of myotonic manifestations. On the spondylograms, and with the help of REG and EMG, the signs typical for the natal damage of the cervical area of the spine and spinal cord were revealed. It is concluded that in most of the cases myotonia arises as a result of natally caused ischemia of the reticular formation of the brain stem.

Birth Injuries↗