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Biomedical subjects

A Joós

Publications and source records attributed to A Joós.

5 recordsLinked to original sources
PubMed1992

[Studying the role of Helicobacter pylori infection in recurrent abdominal pain in children].

The authors studied the role of Helicobacter pylori at recurrent abdominal pain in childhood. Helicobacter pylori infection hasn't been found at the 42 examined children. The endoscopy showed esophagitis in 34 cases. The quick urease test, the histological examination, and the bacterial culture are proposed to carry out in ulcus duodeni, gastritis typ. B ulcus ventriculi and not necessary to carry out if the endoscopy shows only esophagitis--emphasize the authors.

Abdominal Pain↗
PubMed1979

[Condyloma acuminata].

A review of the clinical features and therapeutic possibilities of condyloma acuminatum is presented on the ground of the ten-year experience of the authors. A giant condyloma simplex with benign clinical behaviour and typical histologic pattern and a case of epithelioma starting from a condyloma simplex and leading to the amputation of the penis are described.

Adult↗
PubMed1978

[Endoscopic stomach polypectomy].

During 33 months in 45 patients 52 times an endoscopic polypectomy was performed in the stomach or in the duodenum. The polyp or the polyposis of the stomach in every case should be clarified with the help of endoscopic polypectomy and histological examination. The histological type of the polyps got in toto determines the therapeutic methods essentially deviating from each other. After a polypectomy a regular gastroscopic control is necessary. The 10% frequency of postoperative bleedings after endoscopic polypectomy may be reduced by improvement of the technique.

Gastroscopy↗
PubMed1977

Lesions caused by copper depletion in the chicken. Light and electron microscopic study.

Lesions of the aorta and other organs of copper depleted layers, roosters, day-old chicks and chick embryos were studied by light- and electron microscopy. Copper deficiency results in late and abnormal development of elastic fibres: inhibition of the formation of cross linkages causes a decline of elastin synthesis. The microfibrils constituting the blackbone of the elastic fibre form a honey-comb pattern. The small amount of amorphous elastin is digestible by elastase, the microfibrils are resistant to the action of the enzyme. The aortic elastic fibres are irregularly shaped, broken revealing a honey-comb like pattern. They maintain an increased affinity to PTA. Tears--dissecting aneurysms--arise in the media, vacuoles appear in the heart muscle, haemorrhages and inflammatory changes may arise in other organs.

Animals↗