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A Klisnick

Publications and source records attributed to A Klisnick.

22 records · Page 2Linked to original sources
PubMed1995

[Cervical spinal cord compression caused by ossification of the posterior vertebral ligament. Apropos of a case].

Progressive cervical myelopathy due to an ossification of the posterior longitudinal ligament (OPLL) appeared in a sicilian women. Diagnosis was made by lateral conventional tomograms and CT Scan with mielography. Corporectomy of the 5th vertebra and ablation of the ligament dramatically improved the clinical symptoms. If OPLL seems a common cause of cervical myelopathy in Japan, it is very rate in Caucasian people. Instead of the origin of the patients, progressive tetraparesis of the 60 years old people is the usual presentation of this affection. The cause of OPLL remains unknown: genetic factors, vitamin A, association with diffuse idiopathic skeletal hyperostosis, calcium metabolism abnormalities are discussed. As far as surgical treatment is concerned, choice between anterior and posterior decompression is clarified on the basis of the type and the extension of OPLL.

Adult↗
PubMedDate not supplied

[Rotavirus infection associated with adult thrombotic microangiopathy].

BACKGROUND: A preceding viral syndrome is classically cited as a triggering factor for thrombotic microangiopathy although only anecdotal reports of the causal agent have been documented. CASE REPORT: An 84-year-old woman developed thrombotic microangiopathy secondary to diarrhea. An exhaustive etiological work-up documented the presence of Rotavirus alone in the fecal matter. The clinical course was favorable after 9 plasma exchanges using frozen fresh plasma and was associated with disappearance of the Rotavirus. DISCUSSION: Enterohemorrhagic Escherichia coli is the most commonly identified infectious agent causing thrombotic microangiopathy. A small number of cases caused by viral agents (particularly retroviruses) have also been documented. To our knowledge, this is the first case attributable to a Rotavirus. In our case, the presence of the Rotavirus in the fecal matter and the favorable clinical course when the virus disappeared would be in favor of its causal role in the pathogenesis of thrombotic micorangiopathy.

Aged↗