Effects on insulin output and on pancreatic blood flow of exogenous insulin infusion into an in situ isolated portion of the pancreas.
Explore the source record for details and available documents.
Animals↗
Biomedical subjects
A M Rappaport
Publications and source records attributed to A M Rappaport.
Effects of hormones and of blood flow on insulin output of isolated pancreas in situ.
Explore the source record for details and available documents.
Animals↗
Quantitative determination of insulin output following an intravenous glucose tolerance test in the dog.
Explore the source record for details and available documents.
Animals↗
Effects of hepatic artery ligation on survival and metabolism of depancreatized dogs.
Explore the source record for details and available documents.
Animals↗
[Subcutaneous pediculated autograft of an isolated pancreatic remnant for temporary block of internal pancreatic secretion in dogs].
Explore the source record for details and available documents.
Animals↗
A pedunculated subcutaneous autotransplant of an isolated pancreas remnant, for the temporary deprivation of internal pancreatic secretion in the dog.
Explore the source record for details and available documents.
Animals↗
Autotransplantation of the pancreas.
Explore the source record for details and available documents.
Animals↗
Normal and pathologic microcirculation of the living mammalian liver.
Explore the source record for details and available documents.
Animals↗
Effects of sudden deprivation and restoration of insulin secretion on glucose metabolism in dogs.
Explore the source record for details and available documents.
Animals↗
The effect of mouse hepatitis virus infection on the microcirculation of the liver.
Mouse hepatitis virus type 3 infection results in strain-dependent liver disease. The effects of mouse hepatitis virus type 3 on the microcirculation of the liver in both fully susceptible (Balb/cJ) and fully resistant (A/J) mice were studied. In Balb/cJ mice, 6 to 12 hr following infection, abnormalities in liver blood flow were observed which consisted of granular blood flow in both terminal hepatic and terminal portal venules. In addition, sinusoidal microthrombi were present predominantly in periportal areas. By 24 to 48 hr, liver cell edema and small focal lesions were prominent. At 48 hr, thrombi and hepatocellular necrosis were widespread, and blood was shunted from damaged areas into patent sinusoids. In sharp contrast to these abnormal findings, normal streamlined blood flow was present in the resistant A/J animals at all time points following infection. Since large amounts of virus were demonstrated by immunofluorescene in and by recovery and growth from livers of both resistant and susceptible strains, the presence of the virus per se cannot explain the abnormalities observed.
Animals↗