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Biomedical subjects

A P Torgunakov

Publications and source records attributed to A P Torgunakov.

At least 19 recordsLinked to original sources
PubMed1992

[Experience with unilateral portalization of the adrenal and renal blood in chronic hepatitis].

An experience with treatment of 46 patients with chronic hepatitis and liver cirrhosis with the help of left-sided renoportal venous anastomosis is described. Unsuccessful therapeutic treatment is an indication to operation. In remote period after operation from 50 to 100% of clinical symptoms disappeared in 80% of the patients. Positive dynamics of the results of biochemical analyses and scanning of the liver was noted. The operation is not indicated in patients with formed cirrhosis, portal hypertension over 240 mm water column, decompensation of the liver functions.

Adrenal Glands↗
PubMed1990

[New approach to surgical treatment of diabetes mellitus].

Results of palliative operations in three patients with diabetes mellitus were analyzed. Operations were directed to a decrease of activity of counterinsular hormones--glucagon, glucocorticosteroids, catecholamines. In one patient with a coexistent chronic hepatitis a positive effect was obtained after left-side renoportal venous anastomosis with ligation of splenic vessels. The ligation of splenic vessels and unilateral adrenalectomy were performed in 2 patients with I and II type diabetes mellitus associated with arterial hypertension. A hypoglycemic and hypotensive result was obtained.

Adult↗
PubMed1989

[Oblique-transverse approach to the extrahepatic bile ducts].

A new access to extrahepatic bile ducts is proposed which keeps the lateral abdominal muscles and intercostal vascular-nervous plexuses intact. The spatial conditions of the access were studied experimentally in 108 corpses. The clinical assessment was tested in 102 operations in comparison with the Kocher access. The oblique-transversal access was found to have advantages from the clinico-physiological viewpoint as well (the development of the postoperative period, restoration of the external respiration function and the function of the abdominal wall muscles).

Abdominal Muscles↗
PubMed1986

[Diagnosis and surgical tactics in stable arterial hypertension complicating nephroptosis].

An experience with the surgical treatment of 87 patients with different forms of stable arterial hypertension--vasorenal, nephrogenic, endocrinous-adrenal--has shown that every 5th patient has associated lesions. For the period from 1982 till 1983 the authors diagnosed hyperplasia of the adrenal in 5 of 22 patients having a stable arterial hypertension. When detecting pathological changes of adrenals in arterial hypertension against the background of nephroptosis it is expedient to perform an appropriate intervention on adrenals in addition to correction of the vasorenal component.

Adrenal Glands↗
PubMed1984

[Diagnosis and treatment of vasorenal hypertension].

The examination and surgical treatment of 175 patients with persistent arterial hypertension were analyzed, 45 of them having a lesion of renal arteries. The author recommends a complex of diagnostic examinations of kidneys, renal arteries and adrenals. In cases with organic alterations in the adrenals surgical procedures on the adrenals adequate to the alterations should be performed along with the operation on renal arteries. In long-standing disease it is expedient to perform adrenalectomy on the same side in addition to reconstruction of the renal arteries.

Adolescent↗
PubMed1983

[Changes in the renin-aldosterone system after unilateral portalization of adrenal and renal blood in patients with arterial hypertension].

Twenty-one patients with severe arterial hypertension were operated on using an original procedure, which consisted in creating a left-side renoportal venous anastomosis and right-side adrenalectomy. A considerable hypotensive effect was noted at both early and late follow-up. A drop in vascular peripheral resistance as a result of decreased blood aldosterone and cortisol levels as well as sodium elimination was found to be the principal mechanism of the hypotensive effect.

Adrenal Glands↗