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Biomedical subjects

A S Eremeeva

Publications and source records attributed to A S Eremeeva.

At least 19 recordsLinked to original sources
PubMed1988

[Iatrogenic pathology in pediatrics].

Literature and original data have been analyzed concerned with iatrogenic diseases in fetuses and children at various time periods, as well as the sequelae due to iatrogenic childhood disturbances.

Female↗
PubMed1988

[Primary cardiomyopathies in children].

Available are two cases of primary cardiomyopathy (congestive and oncocytic, respectively) in 8.5-year-old and 18-day-old girls. Analysis of section material indicates that hypertrophic cardiomyopathy is not a rare finding in newborns with interventricular septal affections. The authors review the discussion opinions on the disease etiology and pathogenesis.

Cardiomyopathies↗
PubMed1986

[Pseudotumor processes in children ( according to material from the Filatov Children's Clinical Hospital)].

Pseudoneoplastic tumors are considered as a clinicoanatomic entity with a characteristic macro- and microscopic pattern, obscure etiology and genesis. The dissection data have demonstrated the forms of pseudoneoplastic processes. Observations of the most rare pseudotumors, such as "inflammatory" pseudotumor and tumor-like myositis ossificans are presented. "Inflammatory" pseudotumors are characterized by the involvement of the immune system cells, increased fibrillogenesis, and intensive osteogenesis in the lungs and the stomach. Cases of pseudoneoplastic myositis ossificans combined with tissue malformation are given particular attention.

Adolescent↗
PubMed1982

[Acute appendicitis in newborn infants].

Twenty-two observations of acute destructive appendicitis in newborns of 4 to 28 days of age are presented. A marked necrotic component of the inflammation with perforation and partial self-amputation is noted. It is assumed that a combination of general and local factors is important in the pathogenesis of acute appendicitis in newborns, with the leading role of systemic factors developing in newborns under conditions of hypoxia or infection: disturbance of the circulation in organs increased permeability of the histohematic barrier, loss of resistance and protective role of the intestinal flora with increasing activity of conditionally-pathogenic gram-negative (endogenous and exogenous) microbial flora.

Acute Disease↗
PubMed1978

[Infusion therapy complications in newborn and nursing infants].

The case histories of 306 babies dying at the age under 6 months were analysed. All the babies were given medicinal and nutrient solutions mainly via the subclavical veins for different diseases. In the process of the infusion therapy complications were observed which were distributed into 5 groups: 1--traumatic injuries, 2--air embolism, 3--thrombosis, 4--diffuse hyalin thrombi predominantly in the lung vessels, 5--acute renal insufficiency (ARI). Formation of the hyalin thrombi appeared to be due to the damaging effect of the administered solutions on the vascular endothelium and changes in the properties of erythrocytes and blood plasma. In the development of ARI some importance is ascribed to infusion overloading of the structurally and functionally immature kidneys of young infants.

Catheterization↗
PubMed1976

[Primary hypertension of the lesser circulation].

The article presents a survey of the literature data and findings of the author's own investigations concerning various aspects of primary pulmonary hypertension (PPH). PPH -- is a polyentiological suffering. As a nosological form it was described in the fifities. There are two forms of PPH: congenital and acquired. In its turn, the congenital form comprises two variants of anomalies of the pulmonary vessels: the retnetion of the fetal structure of the pulmonary arteries and an anomaly at the level of intraorgan vascular anastomoses. Constant morphological characteristics of the two forms of PPH are as follows: endosclerosis of major branches of the pulmonary artery and focal sclerosis of their tunica media; a considerable degree of hypertrophy of the right ventricle usually not observed in other diseases. The acquired PPH may develop as a result of exposure to drugs, in thromboembolism, under high mountain conditions, etc. A question arises, whether it is justifiable to call as "primary" the acquired forms of pulmonary hypertension, since in the majority of cases their secondary nature is evident. Thus, the term "primary pullomonary hypertension" should be applied only to congenital forms of the disease. Possible in order to single out this group of desorders it would be reasonable to devise a more suitable name, for instance, "isolated pulmonary hypertension" which will render a broader sense to this polyetiological suffering.

Child↗