Biomedical subjects

A T Tysvaer

Publications and source records attributed to A T Tysvaer.

15 recordsLinked to original sources

PubMed1992

[Head and neck injuries among Norwegian soccer players. A neurological, electroencephalographic, radiologic and neuropsychological evaluation].

69 active football (soccer) players and 37 former members of the Norwegian national team were included in a neurological and electroencephalographic (EEG) study to investigate the incidence of head injuries, mainly caused by heading the ball. 3% of the active players and 30% of the former players complained of permanent problems such as headache, dizziness, irritability, impaired memory and pain in the neck. 35% of the active players and 32% of former players had from slightly abnormal to abnormal EEG, as against 13 and 11% of respective matched controls. There were fewer definitely abnormal EEG changes among typical headers (10%) than among "non-headers" (27%). The former players were also subjected to cerebral computed tomography (CT), a neuropsychological examination and a radiological examination of the cervical spine. One third of the players were found to have central cerebral atrophy and 81% to have from mild to severe (mostly mild to moderate) neuropsychological impairment. The radiological examination of the cervical spine revealed a significantly higher incidence and degree of degenerative changes than in a matched control group.

Adolescent↗

PubMed1992

Head and neck injuries in soccer. Impact of minor trauma.

Head injuries have been shown to account for between 4 and 22% of soccer injuries. Clinical and neuropsychological investigations of patients with minor head trauma have revealed organic brain damage. 69 active football (soccer) players and 37 former players of the Norwegian national team were included in a neurological and electroencephalographic (EEG) study to investigate the incidence of head injuries mainly caused by heading the ball. 3% of the active and 30% of the former players complained of permanent problems such as headache, dizziness, irritability, impaired memory and neck pain. 35% of the active and 32% of former players had from slightly abnormal to abnormal EEG compared with 13 and 11% of matched controls, respectively. There were fewer definitely abnormal EEG changes among typical 'headers' (10%) than among 'nonheaders' (27%). The former players were also subjected to cerebral computed tomography (CT), a neuropsychological examination and a radiological examination of the cervical spine. One-third of the players were found to have central cerebral atrophy and 81% to have from mild to severe (mostly mild to moderate) neuropsychological impairment. The radiological examination of the cervical spine revealed a significantly higher incidence and degree of degenerative changes than in a matched control group.

Adolescent↗

PubMed1989

Brain damage in former association football players. An evaluation by cerebral computed tomography.

Thirty-three former football players from the National Football Team of Norway were examined by cerebral computer tomography (CT). The CT studies, evaluated for brain atrophy, visually and by linear measurements compared two different normal materials. One third of the players were found to have central cerebral atrophy. It is concluded that the atrophy probably was caused by repeated small head injuries during the football play, mainly in connection with heading the ball.

Adult↗

PubMed1989

Soccer injuries to the brain. A neurologic and electroencephalographic study of former players.

Thirty-seven former football players of the Norwegian national team underwent a neurological and electroencephalographic (EEG) examination to investigate the incidence of head injuries due to heading the ball. Sixteen players complained of protracted and permanent symptoms commonly attributed to the post-concussional syndrome: headache, irritability, dizziness, lack of concentration and impaired memory. A significantly increased incidence of EEG abnormalities was found in players compared with matched controls. The high incidence of EEG changes is probably the result of a cumulative effect due to repeated head traumas.

Adult↗

PubMed1988