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Biomedical subjects

A V Samoĭlenko

Publications and source records attributed to A V Samoĭlenko.

36 records · Page 2Linked to original sources
PubMed1986

[Systemic hemodynamic shifts in the arterial and venous portions of the vascular bed in hypoxia].

A reduction of total peripheral resistance is of a major importance for depressor shifts of arterial pressure in moderate hypoxia. The hypoxic stimulus induces a deficiency of increment in cardiac output shifts as compared with venous return. An increment of venous return and cardiac output suggests that the compensation for the depressor shifts induced with the lack of O2 in air and blood, occurs on account of flow induces of the circulation system.

Animals↗
PubMed1983

[Study of the role of the venous return in pressor changes in the systemic hemodynamics by the means of the automatic regulation of its value].

Feed-back experiments with catecholamine-induced pressor changes of arterial pressure (AP) aided to study possible contribution of the venous return changes in cats. Preliminary recorded changes were reproduced with the aid of electronic control of perfusion pump connected with extracorporeal reservoir and venous portion of vascular bed. The reproduced changes of venous return corresponded to those occurring under action of catecholamines. The same maximal increase of AP (by 50%) could be obtained by means of increasing the venous return either by 1/6 with adrenaline (A) or by 1/10 with noradrenaline (NA). Maintaining of the increased AP involves to a greater extent participation of the above parameters in systemic shifts; in response to A 1/2 and in response to NA 1/3 of the pressor response can be maintained by an increase in the venous return. The quantitative contribution of the vascular component (total peripheral resistance) to pressor shifts of AP under the catecholamines action is greater than that of the cardiac component, particularly in response to NA.

Animals↗
PubMed1981

[Blood flow in the inferior vena cava measured by a Doppler ultrasound flowmeter in chronic experiments on cats].

Supercound Doppler's transducers indwelled in the cat v. cava posterior revealed that the higher blood flow values in chronic experiments as compared to acute ones were due to the effect of the chest suction action on the blood flow. The phasic changes of the venous blood flow occurring at resting and under various effects were related to respiration and heart activity, the respiratory component being more obvious than the cardiac one. A significant correlation existed between the mean value of the blood flow in the v. cava posterior and the respiratory changes of the blood flow amplitude.

Animals↗
PubMed1980

[Changes in systemic hemodynamics caused by intra-arterial and intravenous autoperfusion of small blood volumes].

Changes of general peripheral resistance in response to autoperfusion both prior to and after blockade of alpha- and beta-adrenoreceptors depended on its initial level: at a relatively low initial value autoperfusion increased the vascular resistance whereas at high value decreased it. During maximal shifts of hemodynamic parameters the venous return prevailed over the cardiac output. The pressor effect was practically unrelated to the way of blood administration.

Animals↗
PubMed1979

[Adrenergic mechanisms of changes in the resistance and volume vessels of the small intestine in the presence of a reduced volume of circulating blood].

In humorally isolated small intestine, the 10--20%--decrease in the perfusing blood volume against the background of augmented vascular resistance of the small intestine, induces different shifts of the latter's vascular capacitance. At the blockade of small intestine's beta--adrenoreceptors the decrease does not practically tell on the character of shifts in the intestine's resistance and capacitance while the blockade of alpha--adrenoreceptors abolishes the augmentation of these parameters.

Animals↗
PubMed1979

[Hemodynamic structure of pressor reactions to vasoactive substances].

In cats, similar changes of the blood pressure had different hemodynamic structures under the influence of catecholamines or angiotensin. Adrenalin was able to evoke a pressor response by means to either caridac or vascular factor. In the former case, the increase in the cardiac output was induced both by an increasing blood flow towards the heart and by an additive outflow of the blood from the minor to the major circulation; in the latter case, the increase in the general peripheral resistance entailed a decrease in the cardiac output in spite of the augmentation of the venous return which determined a delay of the blood in the minor circulation. The latter variant of interrelationships among hemodynamic parameters can occur under the influence of noradrenaline as well. A combined similarly rendered participation of the cardiac and the vascular factors in the increasing the blood pressure occurs under the influence of angiotensin and sometimes of noradrenaline. The participation of cardiac output in the pressor response may depend on hemodynamic shifts in the minor circulation which either limit or facilitate, or leave unchanged the rate of increment of the cardiac output.

Angiotensin II↗
PubMedDate not supplied

[Effect of different regimes of the artificial respiration on systemic hemodynamics].

An increase in artificial lung ventilation induced a suppression in arterial and venous portions of the circulation, whereas a limited lung ventilation induced pressor shifts of these parameters. The findings suggest that the value of respiratory volume per a cycle rather than shifts in the minute respiratory volume, affects the values of circulation systemic parameters.

Animals↗