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Biomedical subjects

A del Rio

Publications and source records attributed to A del Rio.

9 recordsLinked to original sources
PubMed1992

Calbindin immunoreactivity in normal human temporal neocortex.

Calbindin immunoreactivity in the temporal neocortex was examined in 4 subjects with no neurological, metabolic or malignant disease. The brains were obtained between 1 and 4 h after death and rapidly fixed by perfusion with 4% paraformaldehyde through the carotid arteries, cut into slabs, cryoprotected and stored at -80 degrees C. Sections of the whole left temporal lobe obtained with a freezing microtome were processed free-floating with a well known monoclonal antibody against calbindin according to the peroxidase-antiperoxidase (PAP) method. Calbindin-immunoreactive (CaBP-ir) neurons were found to be local-circuit neurons (interneurons) mainly distributed in the upper cortical layers (layers I, II and III), and were categorized as small multipolar neurons with ascending dendrites ramifying in the molecular layer, small bitufted cells, pyramid-like cells in layer II, horizontal neurons in the molecular layer, multipolar neurons with long descending dendrites, and large double-bouquet cells, some of them exhibiting a very long dendrite with claw-shaped terminals in layer V. Less than 10% of all CaBP-ir neurons were localized in the remaining cortical layers. Pyramidal cells were only very weakly or not stained at all. In addition, CaBP-ir fibres formed a dense plexus in the molecular layer, and vertical bundles 8-10 microns thick and 500-600 microns long, separated by blank spaces 20-40 microns wide were distributed in layers III and V/VI.(ABSTRACT TRUNCATED AT 250 WORDS)

Aged↗
PubMed1985

Dermatomyositis-like syndrome caused by trichinae. Report of two cases.

We report 2 cases of trichinosis in which the clinical features resembled dermatomyositis. In both cases, however, the diagnosis was established histologically by demonstrating trichina larvae in the patients' muscle biopsies. Indirect immunofluorescence and counterimmunoelectrophoresis techniques were used to confirm the diagnosis serologically.

Adult↗
PubMed1978

Anterograde and retrograde bradycardiac block in the His-Purkinje system. Finding in a patient with acute myocardial infarction.

A patient with an acute inferior myocardial infarction developed a complete atrioventricular block and intermitent periods of atrioventricular conduction with QRS complexes showing right bundle branch block associated with left anterior hemiblock. Recordings of the His bundle electrogram showed that the atrioventricular block was infrahisian and that in periods of resumed atrioventricular conduction, the His-ventricle (H-V) interval was long. Ventricular escape beats showed concealed conduction to the atrioventricular node. Anterograde atrioventricular conduction was always resumed through the left posterior division when the preceding division when the preceding intervals between ventricular escape beats and the atrium (V-A intervals) were shorter than 580 msec. The same phenomenon occurred with right ventricular pacing. A retrograde His potential could be observed. Retrograde conduction of ventricular escape beats and ventricular paced beats was blocked if the H-V interval and the interval between the His bundle and the ventricular paced beat (H-V interval) were long (more than 600 msec and 550 msec, respectively). The existence of an intermittent anterograde and retrograde bradycardiac infrahisian block was inferred from the previously mentioned data; a fixed retrograde atrial nodal block was also present.

Acute Disease↗
PubMed1978

Fascicular block within the His bundle.

The findings in a patient with complete atrioventricular block and normal morphology and duration of the QRS complex are presented. A His bundle electrogram was obtained, which led to the location of the atrioventricular block within the His bundle. A careful review of the electrocardiograms obtained during the seven years preceding the onset of complete atrioventricular block showed a QRS complex with the features of left anterior hemiblock and a progressive impairment of atrioventricular conduction. From these data, we inferred that the different degrees of atrioventricular block and the left anterior hemiblock were caused by lesions within the His bundle involving the fibers destined for the left anterior division of the left branch. After the onset of complete atrioventricular block, with the subsidiary pacemaker located in the His bundle distal to the lesions, the QRS complex became normal, indicating the integrity of the bundle branches and fasciculi. The atrial and proximal His potential intervals and those between distal His and ventricular potentials were normal.

Bundle of His↗
PubMed1977

Bradycardia-dependent interatrial block with retrograde left atrial activation.

A patient with a peculiar interatrial block is reported. The electrocardiogram showed a short PR interval and negative P waves in II, III, and aVF, which were preceded, 0-07 s earlier, by another positive P wave present in the right praecordial leads which were absent in the limb leads. From the study with His bundle electrograms, high right atrial electrograms, and bipolar oesophageal electrocardiograms, it could be proved that atrioventricular, His-Purkinje, and right intra-atrial conduction were normal, and that P waves recorded in limb leads represented left atrial depolarization; whereas the ones in the right praecordial leads corresponded to right atrial activation. The vectorial analysis from both P waves and atrial potentials showed that the left atrium was activated in a retrograde fashion, because of an interatrial block. This block was bradycardia dependent and it disappeared in the cycles shorter than 800 ms.

Aged↗
PubMedDate not supplied

The ascending slope of the apexcardiogram. Its relationship with angiographic determination of left ventricular function.

The relationship between the ascending slope of the apexcardiogram (ACG) and direct determination of left ventricular (LV) performance as assessed angiographically by measurement of the ejection fraction (EF), mean velocity of circumferential fiber shortening (mVcF), mean normalized systolic ejection rate (MNSER), and percentage of the systolic shortening of ventricular long axis (%L), were studied in 40 patients with a wide variety of cardiac diseases. The ascending slope correlated closely and significantly (P less than 0.05) with all of them: (1) EF (r = 0.774), (2) mVcF (r = 0.776), (3) MNSER (r = 0.767) and (4) %L (r = 0.668). In a control group of 10 normal subjects, phi was determined and compared with that obtained in patients with depressed LV function, the difference being statistically significant (P less than 0.01). Other noninvasive indices derived from simultaneous phono-mechano-cardiographic readings were also studied. The calibrated ACG did not correlate with any of the hemodynamic indices; however the calibrated carotidogram did correlate with the peak systolic aortic pressure (r = 0.503). The ratio preejection period/left ventricular ejection time correlated significantly, but less than previously reported with the EF (r = 0.574). We conclude that the value of phi leads to additional evidence supporting the use of quantitative ACG as a noninvasive measure of LV performance in patients with cardiac disease.

Adolescent↗