Biomedical subjects

András Vértes

Publications and source records attributed to András Vértes.

4 recordsLinked to original sources

PubMed2006

[Prevention of cardiovascular diseases: collaborative program].

The so-called Program for preventive collaboration model joins the expertise of specialists and GPs and as such allocates for the effective, cost saving, secure screening of individuals with high and intermediate cardiovascular risk and identifying asymptomatic patients within a big population. Based on SCORE risk assessment GPs define their patients cardiovascular risk. In Gottsegen György Institute of Hungarian Cardiology on patients with intermediate or high risk ankle-brachial doppler index, carotis intima media thickness, artery stiffness, microalbuminuria and left ventricular hypertrophy are diagnosed. Based on the results of non-invasive cardiovascular marker tests a mixed score is elaborated. The applicability of the score is then measured for the further risk assessment of individuals with intermediate or high CV risk. The collaboration of GPs and specialists along with the non-invasive cardiological tests are helpful in the early identification of patients with high cardiovascular risk and when making a decision about drug or non-drug treatments.

Albuminuria↗

PubMed2005

[Noninvasive tests of atherosclerotic burden in asymptomatic people with high cardiovascular risk].

Objective of cardiovascular prevention to reduce the risk of major cardiovascular events and morbidity and prolong the quality of life. Strategy of prevention is identification of high risk individuals, and action to reduce their risk factors levels. Noninvasive tests such as carotis artery duplex scanning, ultrasound-based endothelial function studies, ankle-brachial blood pressure ratios, coronary calcium score by electron-beam CT, and high-sensitivity testing for C-reactive protein offer the potential for directly and indirectly measuring atherosclerosis in asymptomatic people. If a patient is found to have abnormal non-invasive test, this patient can be elevated to a higher risk category.

Ankle↗

PubMed2003

[Correlation of lower extremity Doppler index with flow-mediated vasodilation and the extent of coronary disease].

AIMS: The aim of this study was to examine relationship between resting ankle-brachial pressure index and flow mediated vasodilation, and that of the extent of coronary artery disease. PATIENTS AND METHODS: Authors examined 77 subjects. All had peripheral arterial disease (with ankle-brachial pressure index less than 0.8), and angina pectoris (coronary artery disease verified by coronary angiography). Mean age was 64.6 years (range 41-82 years). Flow mediated (endothelial dependent) vasodilatation was measured for all participiant. RESULTS: Authors stated, that ankle-brachial pressure index significantly correlates with flow mediated vasodilatation and the same connection can be supposed between Doppler index and coronary artery disease's extension.

Adult↗

PubMed2003

[Endothelium-dependent and independent vasodilation in young males with previous myocardial infarction].

INTRODUCTION: Flow-mediated vasodilatation of brachial arteries, a non-invasive parameter of endothelial function, is correlated with cardiovascular risk factors. An impairment of flow-mediated vasodilatation in the brachial artery is related to the extent of coronary artery disease. AIMS: The authors examined the relationship between flow-mediated vasodilatation and coronary artery disease morphology in young patients who had myocardial infarction. PATIENTS AND METHODS: 28 young patients (all men) with myocardial infarction (age 22-40, mean age: 35 years). Coronarography revealed one vessel disease in 16 patients (A group) and multivessel disease in 12 patients (B group). The control group was 14 healthy, young patients (age 18-36 years mean age: 30 years) (C group). The authors examined in all subjects flow-mediated, endothelium-dependent vasodilatation following reactive hyperaemia and nitroglycerin induced (NTG), endothelium-independent vasodilatation in the brachial artery with high resolution ultrasound (Acuson 128 X P/10). RESULTS: Patients after myocardial infarction showed impaired flow-mediated vasodilatation compared to those of controls. (p < 0.01). Flow-mediated vasodilatation was lower in multivessel disease compare to one vessel disease. Nitroglycerin induced similar degrees of vasodilatation in the myocardial infarction and control groups. CONCLUSION: Young patients with myocardial infarction may have impaired endothelium-dependent dilatation and the decrease of flow-mediated vasodilatation is related to the angiographic extent of coronary artery disease.

Adult↗