Biomedical subjects

André Luiz Felix Rodacki

Publications and source records attributed to André Luiz Felix Rodacki.

2 recordsLinked to original sources

PubMed2005

Body mass as a factor in stature change.

BACKGROUND: Back pain is a common condition which has been described as a serious public health problem. Spinal shrinkage has been used as an index of spinal loading in a range of tasks. Epidemiological evidence shows that body mass index (BMI: 30 kg/m(2)) is related to the development of low back pain however, no studies have described the stature change patterns of obese individuals. This study aimed to compare changes in stature after an exercise task in obese and non-obese individuals. METHODS: Twenty volunteers were divided into two equal groups; obese: BMI>30 kg/m(2), non-obese: BMI<25 kg/m(2). Stature was measured at 3 min intervals during a 30 min walking task and a 30 min standing recovery period. Tests were performed on two occasions, once with participants loaded during the walking task (10% body mass) and once unloaded. The influence of obesity and load condition on the magnitude and rate of stature change were compared by a two-way ANOVA: FINDINGS: In both groups the stature loss was greater in the loaded than unloaded condition (mean (SD)) (6.52 (1.45)mm and 3.55 (0.93)mm non-obese; 8.49 (1.75)mm and 7.02 (1.32)mm obese: P=0.016). The obese presented a greater reduction in stature in both task conditions. The obese group were unable to recover stature regardless of the task condition during the recovery period (loaded: 0.06 (0.3)mm; unloaded: 0.32 (0.6)mm; P=0.013). INTERPRETATION: It was concluded that the acute response of the spine to loading may represent a risk factor for low back pain in the obese, in addition to the chronic adaptations previously reported. A greater period of recovery may be necessary for obese individuals to re-establish intervertebral disc height. These findings may help to explain the high incidence of back disorders in obese individuals.

Adaptation, Physiological↗

PubMed2002

Vertical jump coordination: fatigue effects.

PURPOSE: The aim of this study was to investigate the segmental coordination of vertical jumps under fatigue of the knee extensor and flexor muscles. METHODS: Eleven healthy and active subjects performed maximal vertical jumps with and without fatigue, which was imposed by requesting the subjects to extend/flex their knees continuously in a weight machine, until they could not lift a load corresponding to approximately 50% of their body weight. Knee extensor and flexor isokinetic peak torques were also measured before and after fatigue. Video, ground reaction forces, and electromyographic data were collected simultaneously and used to provide several variables of the jumps. RESULTS: Fatiguing the knee flexor muscles did not reduce the height of the jumps or induce changes in the kinematic, kinetic, and electromyographic profiles. Knee extensor fatigue caused the subjects to adjust several variables of the movement, in which the peak joint angular velocity, peak joint net moment, and power around the knee were reduced and occurred earlier in comparison with the nonfatigued jumps. The electromyographic data analyses indicated that the countermovement jumps were performed similarly, i.e., a single strategy was used, irrespective of which muscle group (extensor or flexors) or the changes imposed on the muscle force-generating characteristics (fatigue or nonfatigue). The subjects executed the movements as if they scaled a robust template motor program, which guided the movement execution in all jump conditions. It was speculated that training programs designed to improve jump height performance should avoid severe fatigue levels, which may cause the subjects to learn and adopt a nonoptimal and nonspecific coordination solution. CONCLUSION: It was suggested that the neural input used in the fatigued condition did not constitute an optimal solution and may have played a role in decreasing maximal jump height achievement.

Adult↗