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Biomedical subjects

B A Buletsa

Publications and source records attributed to B A Buletsa.

At least 19 recordsLinked to original sources
PubMed1991

[Clinico-epidemiological characteristics of transient cerebral ischemia in the region of the Ukrainian Carpathian Mountains].

Altogether 225 patients with transitory derangements of cerebral circulation (TDCC) underwent clinico-laboratory examinations. Arterial hypertension and cerebral atherosclerosis turned out to be among most frequently occurring factors of TDCC. The provoking factors included psychoemotional stress and drops of barometric pressure. The course and outcome of TDCC were greatly affected by an increase in blood concentration of catecholamines and thyroid hormones. The high concentration of triiodothyronine in the blood contributed to the development of cerebral stroke.

Adult↗
PubMed1990

[Variants of the clinical course of cerebrovascular diseases in various biogeochemical regions of the Ukrainian Carpathian Mountains].

A total of 417 patients with cerebral circulatory disorders were examined in two biogeochemical regions of the UkrainianCarpathian Mountains. The first one is a region with iodine insufficiency; the second one is a region where the people use table salt in excess. It has been established that iodine insufficiency and excessive iodine concentration in the body of man are risk factors of the development of cerebrovascular disease. Besides, in persons with abnormal iodine metabolism, cerebrovascular diseases run a graver course than in those with normal content in the body of these trace elements.

Adult↗
PubMed1990

[Familial predisposition to stroke in hypertension].

Upon examination of 838 hypertensive subjects who had brain insult two groups of patients were distinguished: those whose parents suffered from hypertension and those without familial history of hypertension. It is found that when hypertension is inherited from mother, brain stroke occurs more frequently and in sons rather than in daughters.

Adult↗
PubMed1990

[Neurologic syndromes in calcinosis of the vertebral artery].

Twenty-five patients with calcification of both vertebral arteries were examined. The following neurological syndromes associated with this pathology were revealed: paroxysmal and permanent vegetovascular dystonias, asthenoneurotic, convulsive, cochleovestibular and spinal myelopathy associated with lateral amyotrophic sclerosis.

Adult↗
PubMed1988

[Epidemiology of stroke in the Ukrainian and Slovak Carpathian Mountains].

Different aspects of epidemiology of stroke were studied in residents of 2 adjacent geographic zones: USSR and Eastern Slovak Carpathian. A total of 385 cerebral stroke patients were studied in the USSR and 345 in Eastern Slovakia. Risk factors were determined. Cerebral serial angiography revealed that carotid and subclavian arteries pathology are major causes of stroke.

Adult↗
PubMed1986

[Neuroendocrine syndromes in cerebrovascular pathology].

Study of hypothalamo-hypophyseo-adrenal, hypothalamo-hypophyseo-thyroid, and hypothalamo-hypophyseo-gonadotropic functions in 156 patients with various types of cerebral circulation disorders has permitted identification of 8 neuroendocrine syndromes in the patients with cerebrovascular pathology. The course and outcomes of these disorders in male and female patients with sexual gland dysfunctions are described. The authors believe that dysfunction of any component of the endocrine system may promote cardiovascular decompensation.

Adult↗
PubMed1985

[Several possible pathogenetic mechanisms of cerebral circulatory disorders].

The author examined the function of the hypothalamic-pituitary-thyroid system (HPTS) in 618 patients with various forms of cerebral circulation disorders and also carried out experiments on 50 rabbits. The results of the clinical-laboratory and experimental studies elicited the role of the HPTS in the pathogenesis of cerebral circulation disorders. HPTS dysfunction was shown to impair the thyroid homeostasis which led to a reduction in the cerebral redox processes, imbalance between the coagulation and anticoagulation systems, and changes in the composition of blood trace elements. All these changes contribute to the development of cerebral circulation disorders.

Adult↗
PubMed1978

[Sulfur-containing substances in the urine and blood of patients with cerebral circulatory disorders].

In 138 patients (87 males and 51 females) with hypertensive disease (90 cases) and atherosclerosis (48 cases) the authors studied the content of sulfur and its fractions in the blood and the level of excretion with urine in disturbances of cerebral circulation. In hypertensive disease and cerebral atherosclerosis, complicated by disturbances of cerebral circulation, there were quantitative changes in the level of sulfur-containing substances both in the blood serum and urine. Disorders in the sulfur metabolism indicate a drop of the autotoxic function in the liver, quite possibly related to the influence of pathological changes proceeding in the central nervous system.

Acute Disease↗
PubMed1978

[Role of thyroid gland dysfunction in the pathogenesis and course of cerebral circulatory disorders].

The functional state of the thyroid gland was studied in 160 patients with different forms of disturbed cerebral circulation. The observations included studies of the iodine content and its fractions in the blood serum and a scanning of the thyroid gland. The author established the significance of thyroid gland dysfunction in the pathogenesis of hypertensive disease and cerebro-vascular insufficiency. In patients with hypertensive crises these studies detected the existence of a hyperthyroid syndrome. It was also found that in patients with cerebrovascular insufficiency accompanied by thyroid gland dysfunctions the clinical picture becomes more aggravated.

Cerebral Hemorrhage↗