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B Collaert

Publications and source records attributed to B Collaert.

24 records · Page 2Linked to original sources

Dentine hypersensitivity: a review.

Dentine hypersensitivity is a relatively common pain condition. Local pulpal inflammation, traumatic oral hygiene and dietary habits have been considered in the etiology of dentine hypersensitivity. Dentine hypersensitivity is probably caused by a change in fluid flow in the dentinal tubules, which in turn excites the nerve endings located at the pulp-dentine border. Consequently, treatment success would depend on the prevention of fluid flow across the dentinal tubules (sodium fluoride, stannous fluoride, sodium monofluorophosphate, strontium chloride, sodium citrate, potassium oxalate, and resins/adhesives), or on the desensitization of the nerve endings located at the pulp-dentine border (potassium nitrate). Although a wide variety of treatment methods have been available, clinical data is characterized with ambiguity, warranting further research of the factors which could possibly be implicated in the occurrence of dentine hypersensitivity.

Citrates↗

[The clinical effects of Plax remains to be demonstrated].

Experimental data concerning the plaque removal capability of the prebrushing rinse Plax tend to show only a limited if any effect on dental plaque accumulation. Its effect on gingivitis has only been studied sporadically, but it seems that Plax is not better than placebo in reducing or preventing the amount of gingivitis.

Benzoates↗

[The treatment of dentin hypersensitivity].

Dentine hypersensitivity is caused by a change in fluid flow in the dentinal tubules, which excites nerve endings located in the dentinal tubules and at the pulp-dentine border area. Traumatic oral hygiene procedures, excessive use of acid containing dietary fluids and certain dental treatments have been implicated in the occurrence of dentine hypersensitivity. Initial treatment relies on a positive doctor-patient relationship and the use of a desensitizing toothpaste containing strontium chloride, potassium nitrate or sodium citrate. If no relief is obtained, further treatment attempts need to be undertaken by the dentist--application of fluoride varnishes, potassium oxalate, resins or adhesives.

Citrates↗

[The periodontium as peri-implant tissue; a review].

The periodontium around implants shows similarities as well as differences with that of natural periodontal tissues. Periodontal complications around osseointegrated implants vary from gingivitis and gingival hyperplasia to bone resorption which may finally cause the loss of the implant. Those complications are associated with plaque accumulation. Their treatment is similar as for natural teeth affected by periodontal disease. The bacterial flora of peri-implant pockets does not seem to be different from the flora around natural teeth. Many questions concerning the incidence and severity of peri-implant disease remain to be answered.

Alveolar Bone Loss↗

[Beagle dogs in periodontal research: a review].

This review intends to focus on the role dogs play in periodontal research. Dogs have given insight in the occurrence, the etiology, the pathogenesis and the treatment of periodontal diseases although some differences with humans exist. The occurrence of root resorption, the better healing capacity and the progression rate of periodontitis are among those differences. Knowledge of the periodontal conditions in dogs is required to be able to evaluate the obtained results and its implications for humans carefully. This seems especially true for the ongoing research in tissue regeneration.

Animals↗