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Biomedical subjects

B Glisić

Publications and source records attributed to B Glisić.

15 recordsLinked to original sources
PubMed2002

Mesiodistal size of deciduous teeth in subjects with unilateral cleft lip and palate.

The aim of this study was to investigate whether there was an association between presence of a cleft and reduced size of deciduous tooth on the cleft side. In this investigation, 58 study models of patients with unilateral cleft lip and palate with deciduous dentition were analyzed. The largest mesiodistal dimension of deciduous teeth was measured by a sliding caliper. The size of the same teeth on the side of the cleft and on the non-cleft side was compared. The results of the investigation were statistically analyzed. A difference between the genders was found. In the sample of boys the lateral deciduous incisor and the second deciduous molars on the cleft side were smaller than those on the non-cleft side, while in girls the central and lateral deciduous incisors were smaller on the cleft side than on the non-cleft side. It was concluded that the presence of a unilateral cleft lip and palate was associated with smaller tooth size.

Child, Preschool↗
PubMed1997

Mandibular growth pattern in Turner's syndrome.

In a group of 15 women with 45,X chromosome constitution, mandibular growth type was investigated by using both linear and angular measurements. The sum of the saddle, articular and gonial angle, lower gonial angle and y-axis was significantly greater. In addition the posterior-anterior facial height ratio in women with Turner's syndrome was significantly smaller than in the controls (61 women with 46,XX chromosome constitution), indicating a tendency to backward and downward growth changes in the mandible, caused by an X chromosome deficiency.

Adult↗
PubMed1993

Mesiodistal dimension of permanent teeth in men with Klinefelter's syndrome.

The effects of an extra chromosome on tooth size were investigated and compared to male and female controls, respectively. Mesiodistal dimensions in men with Klinefelter's syndrome were generally found to be decreased when compared to the male controls. A more pronounced inhibitory effect of an extra X chromosome was observed in the case of incisors and canines, indicating a varied influence on this chromosomal anomaly on dental growth.

Adult↗
PubMed1991

[A study of sagittal jaw relationships in patients with Klinefelter's syndrome].

Lateral cephalometric radiographs were used to study sagittal jaw relationships in 22 cases with Klinefelter's syndrome. The following angles were measured: SNA, SNB and ANB. Depending on the size of SNA and SNB angles, all cases investigated were classified in the following groups: bimaxillary prognathism, bimaxillary orthognathism or retrognathism; maxillary prognathism combined with mandibular orthognathism; maxillary orthognathism combined with mandibular prognathism or retrognathism; maxillary retrognathism combined with mandibular prognathism, orthognathism, or retrognathism. The mean value of the ANB angle of the whole group was -0.20 degrees indicating the skeletal class III jaw relationship. Out of 22 cases investigated, 45.4% showed bimaxillary prognathism, and 27.3% had a combination of the maxillary orthognathism and mandibular prognathism. 27.3% of all cases investigated belonged to the remaining groups.

Cephalometry↗
PubMed1990

[Variations of sagittal skeletal relationships in cases with normal occlusion].

Cephalometric radiographs of a sample of 92 cases with normal occlusion were used to study variations in the sagittal jaw relationship. The Schwarz's and Steiner's cephalometric methods of analysis were employed. Results obtained by the Schwarz's method showed that 58.7% of cases had Class I, 16.3% Class II and 25% Class III skeletal relationship. On the other hand, the Steiner's method demonstrated Class I skeletal relationship in 59.8% cases, Class II skeletal relationship in 8.7% of cases and Class III skeletal relationship in 31.5% of cases. Both cephalometric methods indicated Class I skeletal relationship in 39.1% of all cases investigated. In addition, both methods showed the same skeletal relationship (Class I, Class II or Class III) in 58.6% of all cases investigated.

Cephalometry↗
PubMedDate not supplied

[Piezoelectric effect on bone and its origin].

The epidemiological data all over the world show that the reduction of the residual ridge (RRG) of the maxilla and the mandible is one of the major problems in edentulous patients. As it appears after teeth extraction in various degrees and in patients with and without the mobile teeth substitutes, regardless their health condition it can be concluded that the main factor of RRG is the absence of the normal function of the existing teeth. The knowledge about the transformation of the mechanical energy into the electric one by some tissue--especially of the bone--wade, in fact, a new basis for a better understanding of the RRG process. Discovery of electro-mechanical occurrences in the bone, i.e. of piezzoelectricity and of its biological influence on the bone tissue, proved that the electromagnetic disturbance in bone, related to the absence of the function, we considered as a dominant pathophysiological process in RRG.

Alveolar Bone Loss↗
PubMedDate not supplied

[Analysis of factors influencing the amount and localisation of residual ridge reduction of mandible].

The objective of this study was to determine, on a certain number of edentulous patients, the correlation between some parameters of residual ridge reduction (RRR)--(age of patients; parodontopathy as cause of teeth extraction; period of edentulousness; period of denture wearing and reasons of renewed dentures) and classes and sub-classes of RRG in the frontal and molar regions. The aim of these comparisons was to precisely determine the possible presence of loccus minoris of reduction in relation to the studied zones and the mentioned factors of the loss of residual ridge (RR). The study concerned 35 lateral head films of edentulous patients chosen at random. By intersection of data and by their statistical processing, it was established that all reduction parameters, except the age of patients, were important factors or co-factors of this process. They had no specific effect RR. The finding that the molar region was more jeopardized by reduction was explained by the fact that the period of edentulousness and denture wearing were in function of the time, due to an unequal rate of teeth extraction, and that they became the true reason of a more severe reduction and the molar region a more susceptible area to this process.

Alveolar Bone Loss↗
PubMedDate not supplied

[Reconstruction of initial dimensions of the lower residual ridge and classification of reduction in vertical direction].

On the basic of Rtg craniometric parameters described in previous works, in this article a graphical reconstruction of initial dimensions of the lower jaw residual ridge, classification of the reduction according to classes and subclasses of its expression in vertical direction and comparison of reduction intensity in the frontal and molar regions of edentulous jaws are presented. The study concerned 35 lateral head films of edentulous patients, chosen at random. The results of the study revealed the reduction of residual ridge (RRG) in all edentulous patients, and according to the degree of its expression it was different in frontal and mollar regions. By statistic processing of certain classes and sub-classes of reduction and by appropriate tests of significance significant differences in the expression of RRG in these regions were ascertained. According to our finding the molar region was more susceptible to these processes. The reported values offer the possibility for more precise prosthetic reconstruction of the lost tissue and relations in the treatment of edentulous patients.

Alveolar Bone Loss↗
PubMedDate not supplied

[The antiphospholipid syndrome in systemic connective tissue diseases].

The aim of the study was to prove the incidence of antiphospholipid syndrome (APS) in systemic connective tissue disease (SCTD), especially in systemic lupus erythematosus (SLE) and to find out serologic markers of APS. Of 94 patients with SCTD followed up in the last three years 80 had SLE, 7 Sjogren's syndrome, 3 systemic vasculitis, 2 combined systemic connective tissue disease, 1 rheumatoid arthritis, 1 Sjogren's syndrome with vasculitis. Antiphospholipid antibodies (ApSt) were determined by ELISA method. In 31 of 80 patients with SLE (38.07%) antiphospholipid antibodies were proved. In lo of 31 (32.26%) signs of systemic lupus or the presence of APS markers have not been found, while 21 of 31 (67.74%) had some of clinical manifestations of APS (most commonly thrombocytopenia or vascular thrombosis) with evolution of the basic disease. Among our patients with SLE the most common finding was IgG antibody isotype-in 14 or 66.66% of cases. In 5 of 7 patients with Sjogren's syndrome ApAt were proved without clinical evolution of the basic disease and APS. Only 2 of 7 patients of this group showed, together with more increased ApAt, mild evolution of the basic disease and manifestation of APS (livado reticularis and recurrent venous thrombosis).

Antiphospholipid Syndrome↗
PubMedDate not supplied

[Cyclosporin A in the treatment of Behçet's disease].

The results of Cyclosporin A treatment of 15 patients with Behçet's disease were examined in the study--9 females (aged from 32 to 34 years, mean 33 years) and 12 males (aged from 25 to 55 years, approximately 34.33 years). Among them, the majority were with chorioretinitis (10/15 or 66.6%). Besides chorioretinitis two patients were simultaneously with iridocyclltis, while in three the whole nervous system was affected (meningoencephalitis, simultaneously pyramidal syndrome with the signs of extrapyramidal lesion and consciousness disorder in one patient). In one patient myocarditis was revealed, and the one was with deep aphtous ulcer. The approximate daily dose was 5 mg/kg in 13 patients, and 7.5 mg/kg/d in 2 patients. For that, the level of the drug in blood elevated from 79 to 380 ng/ml, approximately 170 ng/ml. The effect was favourable in 12 patients (complete restraint of the disease evolution), partial in 2 patients (slowing of the disease course) while it was incomplete in one patient for the interruption of the therapy 15 days after its beginning.

Adult↗
PubMedDate not supplied

Antiphospholipid syndrome in systemic connective tissue diseases.

Clinical manifestation and immunoserological features of secondary antiphospholipid syndrome (SAPS) were analyzed in this paper in 107 patients with systemic connective tissue diseases. In the group of patients with confirmed systemic lupus erythematosus (SLE), antiphospholipid antibodies (aPL) were positive in 43/93 (46.23%), while in 50/93 (53.76%) they were negative. Among aPL positive patients, 33/43 (76.74%) had clinical manifestations of SAPS, while 10 patients (23.26%) were without any clinical manifestations. The most frequent manifestations of SAPS associated with SLE were: arteriovenous thrombosis in 20/43 (46.51%), thrombocitopenia in 15/43 (34.88%) and autoimmune hemolytic anemia in 7/43 (16.27%). In our patients, rare manifestations of SAPS associated with SLE were recurrent fetal loss (1 case), livedo reticularis (1 case), transversal myelitis (2 cases), neuropathy (2 cases) and aseptic endocarditis (Libman-Sacks) (5 cases). Among 7 patients, with Sjögren's syndrome, clinically manifested SAPS was observed in 2, while in other 5 only increased aPL levels were found, as well as in patients with systemic vasculitis-3, MCTD-2 and Sy. Sjögren with vasculitis-1. One RA patient had thrombosis of v. cava inferior. In majority of patients with clinically present SAPS the levels of both examined immunoglobulin isotypes (IgG + IgM) were decreased-21/40 or 52.5%, while isolated increase of IgG was found in 14 (35%) and isolated increase of IgM in 5 (19.22%) patients. In three out of five patients with Libman-Sacks only LA test was positive. This investigation have shown that arterial and venous thromboses are the most common manifestations of SAPS in SLE. Every blood vessel may be involved (from arteriolae to the aorta and from postcapilar venules to the v. cava).

Antibodies, Antiphospholipid↗