Wanted--a pacemaker lexicon.
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Biomedical subjects
Publications and source records attributed to B H Boal.
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Serial gated radionuclide angiocardiography was used to demonstrate partial reversal of alcoholic cardiomyopathy, following abstention from alcohol. Six months subsequent to total abstention, the resting ejection fraction, which is a sensitive index of left ventricular function, increased from 19 percent to 42 percent. Thirteen months following total abstention, the resting ejection fraction was preserved at 40 percent. During stress, the ejection fraction increased to 53 percent. The clinical implication of this case report is that gated radionuclide angiocardiography may be used to noninvasively evaluate accurately the subsequent course of reversible alcoholic cardiomyopathy.
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The pacemaker syndrome is an iatrogenic disorder that can result from hemodynamic sequelae of ventricular pacing. Symptoms range from fatigability to syncope and occur during the time the ventricles are being stimulated by the pulse generator. Postulated mechanisms include loss of atrioventricular synchrony, vasodepressor reflexes, and retrograde atrial activation. Prevention is attempted by selection of the appropriate pacing mode for the individual patient. Remission results from restoration of atrioventricular synchrony.
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A patient with sick sinus syndrome (SSS) presented with episodic lightheadedness and weakness. The electrocardiographic features were marked supraventricular bradyarrhythmias and paroxysmal atrial flutter. The symptoms lasted for four years and disappeared with the onset of stable atrial flutter which has persisted for the past seven years. Over the 11-year period of observation, there has been progressive involvement of the His-Purkinje system manifested by the development of left anterior fascicular block, right bundle-branch block, and prolongation of the HV conduction time. The patient has refused pacemaker implantation. In the absence of angina and in the presence of a normal heart size, the etiology of his SSS is postulated to be idiopathic fibrosis of his conduction system.
In 21 patients with and 31 without junctional escape beats, a comparison of the symptoms, revealed that there were no significant differences (p greater than 0.05) in the symptoms between these two groups manifesting a sick sinus syndrome. Thus, the occurrence of junctional escape beats did not provide the anticipated shortening of the asystolic pauses to prevent the occurrence of symptoms related to the period of electrical silence. In 7 patients with an escape junctional rhythm, the long pause was interrupted by the occurrence of a regular junctional rhythm. The pause was not preceded by a supraventricular tachycardia, ruling out physiologic overdrive suppression of the junctional pacemaker. The period of asystole preceding the junctional rhythm was not multiple of the R-R interval of the junctional rhythm. 4 additional patients demonstrated long periods of asystole, uninterrupted by junctional escape beats and at other times exhibited shorter pauses which were terminated by junctional escape beats. These findings can be explained by the presence of a 'junctional arrest' which is analogous to sinoatrial arrest. The phenomenon of 'junctional arrest' may be one tenable explanation to account for the lack of protection by junctional escape activity against the symptoms associated with the sick sinus syndrome.
Chylothorax occurred as a manifestation of severe congestive heart failure resulting from rheumatic mitral stenosis. Following replacement of the mitral valve, chylothorax recurred and was treated by thoracocentesis initially and then resolved during six months of therapy with a diet of medium-chain triglycerides. Ultimately, the patient resumed a regular diet with no further accumulation of pleural fluid.
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