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Biomedical subjects

B Roth

Publications and source records attributed to B Roth.

At least 271 records · Page 15Linked to original sources

[Diagnostic procedure in congenital saccharase-deficiency (author's transl)].

The diagnostic procedure is described in a 20 months old infant suffering from hereditary saccharase-deficiency. As a simple and adequate method the kinetic analysis of the oral disaccharide-tolerance-test can be used. The comparison of the areas under the glucose-concentration-curves in blood after the oral monosaccharide (glucose and fructose) and disaccharide (saccharose)-load, can be used as a measure for activity of saccharase in the intestinal mucosa.

Blood Glucose↗

[Subhepatic abscess after cholecystectomy with coagulation active substance].

4 cases of postoperative subhepatic abscess formation after cholecystectomy are reported, where absorbable hemostatic agents for control of bleeding had been used. The etiology of these complications is discussed. The use of local hemostyptica is strongly disadvised in all possibly bacteriologically contaminated procedures.

Aged↗

[Excretion of D-glucaric-acid in newborns with non-hemolytic hyperbilirubinemia after phenobarbital treatment (author's transl)].

The excretion of D-glucaric-acid during 24 hours in the urine, which is a measure for the activity of microsomal liver enzymes, was determined in 33 newborns. From 22 newborns with non-hemolytic unconjugated hyperbilirubinemia 11 were treated with 7.5 mg phenobarbital per kilogram bodyweight 5 days and afterwards the excretion of D-glucaric-acid was measured. In contrast to the untreated newborns there was a significant decrease of the bilirubin concentration in the serum and a concomitant increase of the D-glucaric acid excretion from 0,052 mumol/day/kg to 0,388 mumol/day/kg (mean values). A negative correlation (r = -0,51) could be calculated between the bilirubin concentration in the serum and the excretion of D-glucaric-acid after treatment with phenobarbital.

Bilirubin↗

Narcolepsy and hypersomnia: review and classification of 642 personally observed cases.

In this paper the author gives a survey and a classification of 642 cases of narcolepsy and hypersomnia which he himself studied in the course of 26 years. 368 cases were classified as narcolepsy, 274 as hypersomnia. The author further classifies narcolepsies according to their etiology, clinical form and pathophysiological mechanisms of origin. Hypersomnias are divided by the author into the symptomatic and the functional groups. According to the author it is useful to distinguish "short cycle hypersomnia", i.e. those with short duration of sleep attacks (hours) and intervals, from "long cycle hypersomnia", i.e. those with long attacks (days or weeks) and intervals. The author goes on to describe different forms of symptomatic and functional hypersomnias, such as idiopathic hypersomnia, neurotic hypersomnia, the Pickwickian syndrome" and its variants as well as different varieties of periodic long cycle hypersomnias. Finally the author makes a brief mention of the syndrome of insufficiency of wakefulness.

Age Factors↗