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Biomedical subjects

B Slosberg

Publications and source records attributed to B Slosberg.

4 recordsLinked to original sources
PubMed1980

Hypertension: spectrum of problems and guidelines for management.

Hypertensive patients may present to the emergency department with one of three general problems: high blood pressure due to labile hypertension, chronic hypertension, accelerated hypertension, or a hypertensive emergency; side effects from antihypertensive drugs; or acute medical or surgical illness whose management may be affected by hypertension or by drugs taken for hypertension. We describe these problems and recommend an approach to each that is appropriate in the emergency department.

Antihypertensive Agents↗
PubMed1977

Comparative analysis of emergency department treatment of patients with chest pain.

A vital function of the emergency physician is separating patients with chest pain who require intensive immediate treatment from those who require minimal care. The care of 701 patients presenting with chest pain in two Baltimore hospitals was evaluated using medical record data and follow-up questionnaires to discharged patients. Twenty per cent were admitted. There was a significant difference between the two hospitals in admission rates. Thirty-five per cent of the discharged patients felt no better two weeks after emergency department discharge. Plans for further research include a study to clarify the difference in admission rates and the characteristics of individuals who remained symptomatic at two weeks.

Angina Pectoris↗
PubMed1976

Hydrogen sulfide intoxication. A case report and discussion of treatment.

The toxicity of hydrogen sulfide is thought to be due primarily to reversible inactivation of the respiratory enzyme, cytochrome oxidase, with resultant inhibition of aerobic metabolism. A patient with severe hydrogen sulfide poisoning and consequent profound metabolic acidosis was treated successfully with nitrites and oxygen. The nitrite-induced methemoglobin, by competitively binding the toxic hydrosulfide anion until detoxified, presumably reactivated and protected cytochrome oxidase and therby aided the patient's recovery by enhancing aerobic metabolism. His rapid recovery adds clinical support to the efficacy of nitrite therapy in sulfide poisoning. Therefore, we recommend that severe cases of sulfide poisoning be treated with nitrite-induced methemoglobinemia in addition to vigorous supportive care.

Accidents, Occupational↗