Suppression of 7,12-dimethylbenz( )anthracene-produced adrenal necrosis by steroids capable of inducing aryl hydrocarbon hydroxylase.
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Adrenal Gland Diseases↗
Biomedical subjects
B Solymoss
Publications and source records attributed to B Solymoss.
The influence of pregnenolone-16 -carbonitrile on hepatic mixed-function oxygenases.
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Animals↗
Protection by spironolactone and oxandrolone against chronic digitoxin or indomethacin intoxication.
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Animals↗
Effect of spironolactone and ethylestrenol on benzo(a)pyrene hydroxylase (EC 1.14.1.1) and other chemical constituents of hepatic microsomes.
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Animals↗
Effect of spironolactone, norboletone, progesterone, hydroxydione and beta-diethylaminoethyl diphenylpropylacetate on the disappearance rate of indometacin from blood.
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Acetates↗
Effect of spironolactone on the distribution and excretion of 203-HgCl2 in the rat.
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Animals↗
Effect of spironolactone and proadifen on 7,12-dimethylbenz(a)anthracene-induced haematologic changes.
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Animals↗
Influence of spironolactone and other steroids on the enzymatic decay and anticoagulant activity of bishydroxycoumarin.
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Animals↗
Independence of antimineralocorticoid and catatoxic effects of various steroids.
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Anabolic Agents↗
The role of electrolyte disturbances and extracellular alkalosis in metabolic cardiac necrosis and the preventive effect of amiloride.
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Acid-Base Equilibrium↗
Effect of various steroids on microsomal aliphatic hydroxylation and N-dealkylation.
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Alkylation↗
Protection by catatoxic steroids against meprobamate.
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Anabolic Agents↗
The influence of spironolactone on its own biotransformation.
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Animals↗
Influence of cyclohexylamine on the adrenergic cardiopathy.
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Acid-Base Equilibrium↗
[Effect of cyclohexylamine on experimental myocardial necrosis and blood flow].
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Amines↗
Increased drug metabolism induced by various steroids and its suppression by SKF 525-A.
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Anesthesia, General↗
Electrocardiographic alterations in metabolic cardiac necrosis and the influence of amiloride on these changes.
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Acid-Base Equilibrium↗
Suppression by nucleic acid- and protein-synthesis inhibitors of drug detoxication induced by spironolactone or ethylestrenol.
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Anesthesia↗