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Biomedical subjects

B T Troost

Publications and source records attributed to B T Troost.

At least 37 records · Page 2Linked to original sources
PubMed1989

Permanent cerebral blindness after cardiac catheterization.

Cerebral blindness is usually a transient event that can complicate cardiac catheterization. The neurologic deficit clears in 1-3 days. We present a patient who developed permanent cerebral blindness after undergoing transfemoral cardiac catheterization. Bilateral hemorrhagic occipitotemporal infarcts were demonstrated on magnetic resonance imaging (MRI). The patient also demonstrated denial of blindness and had transient memory disturbances due to occipitotemporal lobe infarct. The probable source of the emboli was cardiac.

Aged↗
PubMed1989

Intrachiasmal hemorrhage: a cause of delayed post-traumatic blindness.

A 49-year-old man presented with blurred vision 9 days following frontal head trauma. Visual loss progressed to bilateral blindness. Magnetic resonance imaging revealed hemorrhage and swelling within the optic nerves and chiasm. Indirect trauma to the anterior visual pathways may cause delayed blindness due to hemorrhage and edema within these structures.

Blindness↗
PubMed1989

A false-positive edrophonium test in a patient with a brainstem glioma.

We report a photographically documented false-positive edrophonium test in a patient with a histologically verified brainstem glioma. While a positive response to intravenous edrophonium should usually be regarded as confirmatory for myasthenia gravis, the possibility of a false-positive test must be considered in patients with atypical physical findings.

Adult↗
PubMed1989

Nystagmus: a clinical review.

A review of different types of nystagmus is presented. The review includes characteristic features of the history and findings on examination of nystagmus which allow classification. Types of nystagmus reviewed include congenital, acquired pendular, acquired jerk nystagmus, special nystagmus types such as periodic alternating and downbeat, and the related ocular oscillations such as ocular myoclonus and superior oblique myokymia.

Adult↗
PubMed1988

Lithium-induced downbeat nystagmus.

Two patients who were treated with lithium for psychiatric illness developed primary position downbeat nystagmus. Previous reports have suggested that lithium causes this type of nystagmus, but other known causes were present in most cases. Several months after stopping the lithium, one patient had marked resolution, while the second patient had only minimal improvement. Valproate sodium proved to be useful in suppressing the nystagmus in the second patient. Lithium carbonate is a cause of primary position downbeat nystagmus. The nystagmus may be permanent or require several months of abstinence for improvement.

Adult↗
PubMed1987

Facial numbness and dysesthesia. New features of carotid artery dissection.

Facial numbness and dysesthesia have not been emphasized as presenting features in spontaneous internal carotid artery dissection. Progressive facial pain, accompanied by oculosympathetic paresis, altered taste, and facial numbness suggest the possibility of basal skull neoplasm. We describe a patient, with previously undiscovered fibromuscular dysplasia, who presented with severe neck and face pain, dysgeusia, oculosympathetic paresis, and markedly reduced facial sensation due to a spontaneous vascular dissection. Altered facial sensation should now be included in the symptomatology of internal carotid artery dissection.

Aortic Dissection↗
PubMed1986

See-saw nystagmus associated with Chiari malformation.

See-saw nystagmus has been seen with tumors of the parasellar region and diencephalon, brain-stem vascular lesions, syringobulbia, and after trauma. We report see-saw nystagmus in an adult with a Chiari malformation that was diagnosed by magnetic resonance imaging and confirmed intraoperatively. This patient's condition improved after surgical decompression. An association between see-saw nystagmus and Chiari malformation is potentially important because early diagnosis and decompression may improve neurologic function and prevent further deterioration.

Adult↗
PubMed1986

Lateropulsion and upbeat nystagmus are manifestations of central vestibular dysfunction.

An elderly man presented with acute onset of gait instability, characterized by leaning to the left while walking, vertigo, diplopia, and transient facial numbness. The examination was remarkable for ocular lateropulsion and primary position upbeat nystagmus. Computed tomography of the brain revealed a right-hemispheric cerebellar infarction. This report illustrates that lateropulsion can occur in cerebellar lesions, but that it may be contralateral to the cerebellar hemisphere involved. Primary position upbeat nystagmus and lateropulsion may represent forms of central vestibular dysfunction due to interruption of vestibulo-ocular pathways.

Aged↗
PubMed1984

Monocular elevation paresis caused by an ipsilateral lesion.

A 52-year-old woman presented with a paresis of elevation of the right eye, equally severe in abduction and adduction (monocular elevation paresis). CT demonstrated a small, right-sided tumor of the mesodiencephalic junction. Monocular elevation paresis has been attributed to lesions of the contralateral pretectum, although proof has been lacking. Now, with documentation of an ipsilateral brainstem etiology, we can postulate a lesion affecting the upgaze efferents from the ipsilateral rostral interstitial nucleus of the medial longitudinal fasciculus.

Brain Neoplasms↗
PubMed1983

The effects of age on normal saccadic characteristics and their variability.

Age effects on human saccadic eye movements were tested with infrared reflectance oculography in 34 subjects. In contrast to a prior report, only a slight non-significant change was observed in saccadic velocity and duration. An increase in saccadic latency comparable to that found in several previous reports was observed, however. All parameters showed considerable intersubject variability for both age groups. Decreased velocities or increased durations outside of these normal, broad ranges should be regarded as pathological for all subjects; they are not physiological effects of the aging process.

Adolescent↗
PubMed1981

Carotid-cavernous sinus fistula after external ethmoid-sphenoid surgery. Clinical course and management.

The occurrence of a carotid-cavernous sinus fistula following transethmoidal-sphenoid sinus surgery is unusual. The etiology of this complication is related to the variations in anatomic position of the carotid artery and in the thickness of bone overlying the artery in the lateral sinus wall. In the case reported herein, the clinical course was complicated by the development of cranial nerve palsies and ocular ischemia. The patient's symptoms worsened following an attempted to close the fistula with a microcatheter balloon and isobutyl-cyanoacrylate embolization. Dramatic improvement in the patient's condition was obtained by subsequent occlusion of the carotid artery on the side of the fistula with a microcatheter balloon.

Arteriovenous Fistula↗
PubMed1981

Acquired cyclic esotropia in an adult.

A 67-year-old women developed periodic alternate-day esotropia, documented with videotape, 16-mm film, and quantitative eye-movement recordings, after retinal detachment surgery. The eye movements suggested that central reprocessing and adaptation to a peripheral defect was in part responsible for this adult form of acquired cyclic esotropia.

Adult↗