Biomedical subjects

Berry M Van Gelder

Publications and source records attributed to Berry M Van Gelder.

3 recordsLinked to original sources

PubMed2006

Paradoxical atrial undersensing: noise rate reversion or amplifier ringing?

Atrial tachy-arrhythmias may give rise to mode switching or noise rate reversion in dual-chamber pacemakers. In case of high amplitude of the atrial electrogram during tachycardia, a paradoxical behavior of atrial sensitivity programming can be observed. Two patients with implanted dual-chamber pacemakers showed intermittent and complete loss of atrial sensing during atrial tachycardia during device programming to a higher atrial sensitivity setting. This phenomenon is caused by amplifier ringing and can be considered as a state of amplifier saturation, which disables atrial sensing in spite of the high amplitude of the atrial signal. In patients with high amplitude of the atrial electrogram during atrial tachycardia, a paradoxical behavior of sensitivity programming resulting in atrial undersensing can be observed.

Adult↗

PubMed2006

Preservation of ventricular capture by anodal stimulation after dislodgment of an epicardial electrode.

A 58-year-old man had a bipolar epicardial left ventricular (LV) implanted for cardiac resynchronization therapy after a failed transvenous approach. The system was programmed in an LV tip-right ventricular coil configuration, but during follow-up loss of LV capture with recurrence of symptoms occurred. Changing to an LV tip-LV ring configuration restored LV pacing through anodal capture. Loss of cathodal capture was caused by dislodgment of the cathodal electrode due to a broken fixation suture. Anodal capture was used to reinstall resynchronization therapy, which resulted in clinical improvement. There were no adverse effects from anodal stimulation in a follow-up period of 6 months.

Bundle-Branch Block↗

PubMed2003

Successful implantation of a coronary sinus lead after stenting of a coronary vein stenosis.

Dislodgment of the coronary sinus lead was observed in a 79-year-old patient 8 months after implantation of a biventricular pacing system. A severe stenosis in the posterolateral branch, in which the lead was previously positioned, prohibited reinsertion of the lead. Because no other branches with adequate anatomy for lead insertion were available in the targeted area, the stenosis was dilated and stented. Subsequently, the left ventricular lead could be reimplanted in the same vessel.

Aged↗