[Psychological approach to obesity in children].
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Biomedical subjects
Publications and source records attributed to C Flavigny.
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This work focuses on the Anglo-Saxon idea concerning "hyperactivity" and "hyperkinesis" and the French-language idea of "child psycho-motor instability". The author's own personal study (having two separate parts, on the one hand studying the psychic functioning of parent and their interaction with their child, and on the other, studying material gathered on the individual psychotherapy of unstable children), goes along with the French school of thought, highlighting the extent of incestuous sexual advances toward children (especially boys) in the family unit and the sexual nature (in the sense of child sexuality) of this excitement as the source of their instability, justifying a comparison between the unstable child and a Don Juan-type of instability. What comes out is epistemological thinking on Anglo-Saxon and French-language ideas, in particular criticism of the pre-suppositions in the Anglo-Saxon way of seeing things, which seems only to envisage the characterization of a syndromic range, rather than an organic etiology, this being more often implicit; distanced by the idea of psychodynamics, which predominate in the French-language studies, integrating the symptom of "psycho-motor instability" in the general "wholeness" of the child and evaluating ways of parent-child interaction.
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This study deals with a case of "ophtalmoplegia plus" with histochemistry and electron microscopy of a muscle biopsy and full pathological examination in a 29 year old woman. Ragged-red fibers with abnormal mitochondria, cerebral spongiosis mostly involving white matter, perimacular pigmentary retinopathy and scattered myocardial fibrosis interrupting the Hiss'bundle were found. A goiter with hypersecretion of thyroid hormones and thyroid and ocular muscles changes characteristics of Graves' disease without any hypermetabolism occurred in her late twenties. This could be related to the absence of response from the abnormal mitochondria to the thyroid hormones.