Biomedical subjects

C Marnay-Gulat

Publications and source records attributed to C Marnay-Gulat.

At least 19 recordsLinked to original sources

PubMed1979

[Vitamin D3 sulfoconjugate distribution and evolution in pregnant or lactating rats].

24 hrs after intravenous injection of vitamin D3 35S sulfoconjugate the radioactivity in kidney is about twice as much in pregnant Rats as in lactating Rats. It decreases from 24 to 48 hrs in pregnant but increases in lactating Rats kidneys. Hydrolysis of vitamin 3H D3 sulfate is detected in kidney and liver extracts, the ratio of free vitamin D3 on vitamin D3 sulfate is higher in pregnant than in lactating animals. Mammary glands of lactating Rats contain mainly the unchanged vitamin D3 sulfate which can be hydrolysed by new born suckling pups.

Animals↗

PubMed1978

Effects of glucocorticoids upon adrenal and urinary epinephrine and norepinephrine and the activity of enzyme phenylethanolamine-N-methyltransferase in rats made partially deficient in vitamin D: role of vitamin D supplementation.

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Adrenal Glands↗

PubMed1978

Effects of cortisol on adrenal phenylethanolamine-N-methyltransferase: antagonistic effects of vitamin D in hypophysectomized rats fed a vitamin D free diet.

The influence of individual or combined administration of vitamin D and hydrocortisone on adrenal phenyl-N-methyltransferase (PNMT) in hypophysectomized rats deprived of vitamin D for 1 month has been studied, as well as adrenal and urinary epinephrine and norepinephrine. Daily administration of vitamin D for 11 days decreased PNMT activity and increased adrenal norepinephrine, whereas adrenal epinephrine remained unaffected. Epinephrine and norepinephrine urinary excretion increased after four and nine injections of vitamin D from the hypophysectomized rats fed a vitamin D free diet for 1 month. Hydrocortisone treatment produced increases in adrenal PNMT and epinephrine as well as in urinary excretion of both monoamines. The effects of individual administration are neutralized when the two products are injected together.

Adrenal Glands↗

PubMed1978

[Effect of thyrocalcitonin and an alpha blocking agent in the secretion of amylase, in vitro, on rat parotid and pancreas].

Porcine Calcitonin, together with an alpha adrenergic blocking agent, stimulates amylase release from rat parotid or pancreas, in vitro. A beta blocking agent, propranolol, inhibits this effect. In pancreas only, the coupling of stimulus by calcitonin and phenoxybenzamine involves calcium control.

Amylases↗

PubMed1978

[Study of cyclic AMP in rat tissue during the phase of hypocalcemic activity of parathyroid hormone].

As soon as two minutes after PTH i.v. injection the blood and kidney AMPc level is maximally increased but the lowering blood calcium effect remained till to ten minutes. At this time the calvaria AMPc level reached its maximum just at the onset of hypercalcemic effect of PTH. Thus AMPc increase in tissues is consistent with the two opposite effects of PTH.

Animals↗

PubMed1977

[Norepinephrine interference in adrenal hormone balance related to blood calcium effects of PTH (author's transl)].

Decreased hypercalcemic effects of PTH are observed after medulloadrenalectomy, (table I), metoprione (table II) or cortisone (table IV) treatments in guinea pigs. 2. Norepinephrine restores the blood calcium effects of PTH in these cases but epinephrine is inactive. 3. Immediate activation of kidney adenylate cyclase by PTH (increased urinary AMPc) is also reduced in medulloadrenalectomised, cortisone or metopirone treated guinea pigs (table V). But in this test, norepinephrine reduces the effect of PTH (fig. 1).

Adrenal Glands↗

PubMed1972