[Crystallographic method of investigations in medicine].
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Copper↗
Biomedical subjects
D B Kalikshtein
Publications and source records attributed to D B Kalikshtein.
[Classification of non-coronarogenic myocardial damage].
A working classification of noncoronarogenic damage to the cardiac muscle is suggested in which 4 general-pathology groups are considered: myocardial dystrophies, myocarditis, cardiomyopathy, and neoplasms. The main principles are presented for grouping diseases of the myocardium not associated with ischemic heart disease, hypertension or rheumatic fever. Certain problems of the clinical picture and differential diagnosis of dystrophic, inflammatory, and idiopathic damages to the heart muscle are discussed.
Atrophy↗
Hypersensitivity of delayed type manifested during the development of allergic lesions of the myocardium.
Changes in humoral and cellular responses were studied during the development of allergic lesions of the myocardium in rabbits. These responses,manifestations of hypersensitivity of delayed type, are connected with transformation of cells of the active mesenchyme, responsible for the process of immuogenesis, into lymhocytes and plasma cells. In allergic lesions of the myocardium hypersensitivity of delayed type is manifested as lympho-marcrophagal reaction in the intermuscular stroma of the heart, the thymus, and lymph glands and also as blast transformation of the lymphocytes. A complex series of disturbances of immunologic homeostasis arises, in which the primary components are micronaglopahies, blast-transformation of the lymphoctes and activation of iysosomal enzymes.
Adjuvants, Immunologic↗
[Myocardial damage syndrome in allergic conditions].
Experimental and clinical studies were conducted that permitted to suggest a possible direct damaging effect of allergy upon the myocardium. Distinct types of allergic reactions of the myocardium were singled out in cases of the states being studied, namely infectious-allergic myocarditis and bronchial asthma. The reactions observed in infectious-allergic myocarditis pertain to hypersensitivity of a delayed type which morphological manifestation is embodied in cellular lymphohistocytic, monocytic perivascular infiltration and myocardial stroma damage. In bronchial asthma haemodynamic disorders are noted in the vessels of the microcirculatory bed, as well as oedema of the stroma and muscle fibers destruction.
Animals↗