Guided tissue regeneration.
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Biomedical subjects
Publications and source records attributed to D B Kirkham.
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A case of a 37-year-old female who had a squamous cell carcinoma on the attached gingiva is discussed. The report emphasizes the importance of a correct diagnosis of all lesions, including lesions that appear atypical. A brief review of the literature also is presented.
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This case report describes the treatment of a patient with periodontosis. Surgical treatment was used on all first molar and maxillary incisor areas. Mucoperiosteal flaps were made, and, at six months, evidence of osseous regeneration and formation of new lamina dura was seen on the radiographs. This example showed that an instance of periodontosis may respond to this type of therapy.
The problem of phenytoin-induced gingival hyperplasia is dicussed within the context of consultative planning between oral care practitioners and the physician managing the epilepsy. The reluctance of the neurologist or of the patient to change an ongoing anti-convulsant program is often a response to a long history of tedious steps in medication adjustments to establish a level of seizure control without sacrificing alertness and mood control. Realistic indicators for discontinuing phenytoin are enumerated and therapeutic alternatives in treating phenytoin hyperplasia are discussed. A complicated case of seziure control is offered as an example of requirements in the medical control of epilepsy and the context in which periodontal therapy can be planned in conjunction with these medication adjustments. The mechanism of phenytoin induction of gingival hyperplasia is briefly discussed. Phenytoin (Dilantin) and a newer agent, valproic acid (Depakene), are compared as alternatives in seizure control.
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One hundred permanent human teeth, extracted because of periodontal disease, were studied to determine the percentage of teeth in which accessory canals were located within a periodontal pocket. Pocket depth and other data were recorded before the extractions. After extraction, the pulpal contents were removed and pulp chambers and canals filled with a radiopaque material by an injection process. The teeth were radiographed from different angles to observe the accessory canals. Ten teeth also were studied histologically to compare the results with those obtained by the injection technique. Several conclusions were drawn from this study. Two percent of the 100 teeth studied had accessory canal located within a periodontal pocket. Seventeen percent of the teeth examined had one accessory canal, 6% had two accessory canals, and no teeth had more than two accessory canals. As groups, mandibular premolars and mandibular molars had the highest percentage of teeth exhibiting accessory canals, and maxillary molars and mandibular incisors had the lowest percentage of teeth with accessory canals. The histologic evaluation and the injection technique study showed a direct correlation as to the presence or absence of accessory canals. It was impossible to make any correlation between restorations, caries, attrition, or type of periodontal bone loss and the presence of accessory canals found in periodontal pockets.
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