Biomedical subjects

D Cenić

Publications and source records attributed to D Cenić.

3 recordsLinked to original sources

PubMed1992

Pain and tooth pulp evoked potentials.

The purpose of this study was to verify whether the late components (N 140) of TPEPs were a reliable index of pain intensity. In the group with acute pain, except for higher amplitudes of the second negative wave, the plateau phenomenon has been noted. Analgesic and placebo effects indicated that the method used offers more objective than subjective components, because the real value of amplitudes is about 10% of control values for analgesic and between 60 and 70% for placebo. Shorter latency periods were observed in all parts of evoked potentials in the patients suffering from trigeminal neuralgia, as occurrence of a greater number of waves; while amplitudes on the affected side were significantly higher than on the healthy side.

Dental Pulp↗

PubMed1991

Tooth pulp evoked potentials (TPEPs) in multiple sclerosis patients.

The purpose of this study was to determine possible changes of the tooth pulp evoked potentials in patients with multiple sclerosis, and to compare these with other types of evoked potentials, in the same patients. The results showed TPEP changes in all multiple sclerosis patients. In 12 patients the changes were observed on both sides, while in 6 on one side only. It is interesting to note that the second negative wave was characterized by two peaks. The first peak possessed latency of approximately 125 ms that was significantly shorter than in the control group. The second peak appeared after about 195 ms. The significant prolonged latency of the second negative wave was observed in 100% of TPEPs, 95% VEPs and 72% AEPs. In only one case the changes were observed in TPEPs, while VEPs and AEPs were normal. It could be concluded that TPEPs, as well as other types of evoked potentials, are a sensitive indicator of the demyelination process.

Adolescent↗

PubMed1980

Interaction of pulmonary and systemic hypertension. An experimental study.

The interaction of systemic and pulmonary experimental hypertension was studied in rats exposed to chronic hypoxia and to the Goldblatt procedure both separately and simultaneously. The systemic hypertensive effect of the Goldblatt procedure was unaffected by concomitant hypoxia. Chronic hypoxia caused a similar pulmonary pressor response in non-treated rats and in Goldblatt-hypertensive rats. Pulmonary arterial pressure was found to be elevated in renal hypertensive, nonhypoxic rats. Faced with a similar relative increase in pressure load, the right ventricle is more prone to develop hypertrophy than the left one.

Animals↗