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Biomedical subjects

D G Black

Publications and source records attributed to D G Black.

7 recordsLinked to original sources
PubMed1994

Non-fundholding in Nottingham: a vision of the future.

The 1991 health service reforms introduced the internal market and grave individual fundholding practices budgets with which they could attempt to secure preferential access to secondary health care for their patients. In the view of many doctors this undermined the principle of equity on which the NHS was founded. In Nottingham 200 non-fundholding general practitioners have joined together to act in liaison with their purchasing health authority. A committed representative group of general practitioners can collectively offer more time and knowledge to the contracting process while minimising the impact on clinical workload. As a large purchaser with low management costs the group has secured access to quality secondary care which is equitably available to all patients, preventing the development of a local two tier service. Nottingham's non-fundholding model of commissioning is equitable and efficient.

Family Practice↗
PubMed1984

Effects of treatment for hypertension on cerebral haemorrhage and infarction.

One hundred and sixty nine patients admitted to hospital for stroke over 30 months were examined to see whether treating hypertension had influenced the incidence of cerebral haemorrhage and infarction. Seventy eight (46%) of them had normal blood pressure, 47 (28%) previously diagnosed hypertension for which they were receiving treatment, and 44 (26%) previously undiagnosed and untreated hypertension. Haemorrhagic stroke was commoner among patients with untreated hypertension, whereas infarction was commoner in patients with treated hypertension. Infarction and haemorrhage were equally prevalent in patients with normal blood pressure. Effective treatment in this population seemed to have had a substantially different impact on vascular disease, giving rise to cerebral haemorrhage as opposed to infarction. This is consistent with evidence from other studies that treatment for hypertension has little or no effect on the progression of atheroma.

Aged↗
PubMed1984

Beak and feather dystrophy in wild sulphur-crested cockatoos (Cacatua galerita).

Three flocks of wild sulphur-crested cockatoos in southeastern Australia had a 10-20% prevalence of feather and beak deformities. In affected birds, the crest, tail, contour and down feathers were lost or reduced in length owing to a "pinching off" 0.5-1 cm above the skin. The underlying skin was discolored brown. Flight feathers appeared normal and could sustain flight. In mildly affected birds, only the crest and down feathers were involved. Histological examination of affected feathers showed necrosis of developing cells in their epidermal collars. The epidermis was moderately hyperplastic and contained numerous large macrophages, with large intracytoplasmic granules. Electron microscopic examination showed these granules to consist of regular lattices of 17-20-nm-diameter particles, resembling parvovirus particles. In some birds the upper beak was enlarged and had longitudinal cracks. Histological examination of the beak showed necrosis of its epidermis. It was concluded that a viral infection may have caused feather and beak dystrophy in these birds.

Animals↗
PubMedDate not supplied

Acuaris skrjabini infection of the gizzard of finches.

Adult finches from aviaries in Victoria, Australia, were reported to experience a 4 to 5% mortality rate. Affected birds were lethargic and inappetant and had mild diarrhea for 1 to 2 days before death. Lesions found on post-mortem examination were gizzard mucosal necrosis, with an associated 30 to 60 Acuaria skrjabini spiruroids in each bird and surrounding inflammation. Treatment of remaining finches in the aviaries with either 80 mg of levamisole or 50 mg of fenbendazole per liter of drinking water for 3 days prevented further cases. Acuaria sp. infection can be an important predisposing factor in the mortality of aviary finches.

Animals↗