A new method for determining the distribution of allergenic fractions in biological materials: its applications to grann pollen extracts.
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Adolescent↗
Biomedical subjects
D G Marsh
Publications and source records attributed to D G Marsh.
Studies on "allergoids" prepared from naturally occurring allergens. I. Assay of allergenicity and antigenicity of formalinized rye group I component.
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Allergens↗
Importance of sulfhydryl groups for lipolytic action in the isolated fat cell.
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Adenine Nucleotides↗
In vitro studies of rye grass pollen antigens.
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Allergens↗
Biological studies on free endotoxin and a non-toxic material from culture supernatant fluids of Escherichia coli 078K80.
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Animals↗
Free Endotoxin.
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Bacteriological Techniques↗
Purification and physico-chemical analysis of fractions from the culture supernatant of Escherichia coli O78K80: free endotoxin and a non-toxic fraction.
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Amino Acids↗
Relation of renal arterial pressure to activity of renin-angiotensin system in renal hypertension.
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Angiotensin II↗
Allergens from common rye grass pollen (Lolium perenne). I. Chemical composition and structure.
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Allergens↗
Allergens from common rye grass pollen (Lolium perenne). II. The allergenic determinants and carbohydrate moiety.
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Alkylation↗
The allergenic activity and stability of purified allergens from the pollen of common rye grass (lolium perenne).
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Allergens↗
'Isoallergens' from rye grass pollen.
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Allergens↗
Genetic basis of IgE responsiveness: relevance to the atopic diseases.
Genetic analysis of 170 subjects in 11 extended Amish families revealed evidence for linkage of five markers in chromosome 5q31.1 with a gene controlling total serum IgE levels. No linkage was found between these markers and specific IgE antibody levels. Analysis of total IgE within a subset of 128 IgE-antibody-negative sib pairs confirmed evidence for linkage to 5q31.1, especially IL4 (p = 4 x 10(-6)). These and other data suggest that IL4 or a nearby gene regulates IgE production in a non-antigen-specific (noncognate) fashion and provide evidence for a possible link between asthma and the IL4 gene.
Antibody Formation↗