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Biomedical subjects

D Hilweg

Publications and source records attributed to D Hilweg.

At least 19 recordsLinked to original sources
PubMed1991

[Aortobronchopulmonary fistula following a pulmonary contained aortic rupture in patients with a thoracic aortic aneurysm and severe aortic sclerosis].

Rupture of the thoracic aorta is usually widespread and fatal. Rupture limited by the lung and aorto-bronchopulmonary fistula are rare. We report two patients with a contained perforation of the thoracic aorta, who presented with haemoptysis. Chest radiographs demonstrated an aortic aneurysm and pulmonary haemorrhage in the first, while in the other perforation of an atherosclerotic aorta with a pulmonary haematoma was misinterpreted as bronchial carcinoma. In both cases contrast enhanced CT clearly demonstrated the abnormality in the aorta and the pulmonary haemorrhage. Since aortography often fails to demonstrate a perforation, CT or MRI should be performed early in patients with suspected rupture of the thoracic aorta.

Aged↗
PubMed1990

[Mediastinal and bronchovascular amyloidosis. An unusual x-ray morphologic finding].

The clinical and radiographic aspects of an unusual case of mediastinal and bronchovascular amyloidosis are presented. Besides hilar and mediastinal lymphadenopathy, extensive amyloid deposition in the peribronchial and perivascular connective tissue sheaths can be observed. Plain film radiographs and CT demonstrate an uncommon pattern of increased bronchovascular markings, which is discussed. Because of peribronchial amyloid deposits, bronchoscopy is of no help in demonstrating this special type of tracheobronchial amyloidosis.

Amyloidosis↗
PubMed1989

[Experimental research on the quantitative computed tomographic prediction of the compressive strength of the thoracolumbar vertebrae].

Testing 98 motion segments we investigated the possibility of a prediction of the compressive strength of thoracolumbar vertebrae by QCT. The ultimate compressive strength can be predicted from the density of the trabecular bone and from the size of the endplates--both determined by QCT--with an error of 1 kN. The increase of compressive strength in craniocaudal direction is calculated at approximately 0.3 kN per anatomical level. This variation is due to the increase of the endplate areas.

Adult↗
PubMed1989

Prediction of the compressive strength of human lumbar vertebrae.

The compressive strength of 98 specimens of motion segments of human thoracolumbar spines was measured. In addition, the density of the trabecular bone in the midplane of the vertebrae was assessed by quantitative computed tomography (QCT); the size of the vertebral endplates was measured by CT as well. The results show that the compressive strength of thoracolumbar vertebrae can be predicted from the product of density and end-plate area, with an error of estimate of 1 kN. The data of the experiment allow for an in vivo prediction of the strength of vertebrae to quantify the risk of fracture in physically very demanding tasks, to support expert opinion in trauma cases, or to assist in therapeutic decisions in cases of severe osteoporosis.

Humans↗
PubMed1988

Prediction of the compressive strength of vertebral bodies of the lumbar spine by quantitative computed tomography.

The ultimate compressive strength of 36 thoracolumbar vertebrae was determined experimentally. In addition, the trabecular bone mineral content was measured by single energy quantitative computed tomography. The areas of fractured endplates were also determined by computed tomography. The results show that a linear relationship exists between the compressive strength and the product of bone density and endplate area. These data allow an in vivo prediction of vertebral body strength using a noninvasive method with a standard error of estimate amounting to less than 0.95 kN.

Adult↗
PubMed1976

[Animal experiments on the distribution and excretion of an intravenously injected biliary contrast medium (Ioglycomate) (author's transl)].

The distribution and excretion of an intravenously injected, radioactive biliary contrast medium (Ioglycomate) were studied in the mini-pig after the induction of choledochal, thoracic duct and gall bladder fistulae. The investigations were carried out with free lymph and bile flow during artificial cholestasis and after occluding both ureters. Following intravenous injection of 0.24 to 1.0 ml. Bilivistan, there was a cholersis up to six times original value. Ther thoracic duct lymph showed only a small volume rise. The lymphatic circulation played no significant quantitative role in contrast distribution of Ioglycomate in the animal, nor did it influence the blood clearance curves. During normal bile flow, only 1.3% of the injected dose appeared in the thoracic duct within three hours, and during acute biliary obstruction, only 2.6%. The investigations have indicated a biliary transport maximum Ioglycomate in the mini-pig of 2.9 mg./min./kg. body weight. If the excretory capacity of the liver is exceeded and during cholestasis there is compensatory excretion of the biliary contrast medium through the kidneys.

Animals↗