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Biomedical subjects

D Ia Silin

Publications and source records attributed to D Ia Silin.

5 recordsLinked to original sources
PubMedDate not supplied

[Early transient incapacitation of monkeys exposed to high-energy electrons and gamma-irradiation in supralethal doses].

Irradiation of Macaca fascicularis with electrons (24 MeV, 46 and 104 Gy) and gamma-quanta (60Co, 77 and 138 Gy) caused early transient incapacitation (ETI) and severe symptoms of neurological disturbances. Two types of the ETI clinical patterns were distinguished: a dyskinetic type marked by ataxia; and a severer, comatose type manifested by disappearance of voluntary movements, areactivity, and temporary loss of reflexes.

Animals↗
PubMedDate not supplied

[The electrical activity of the monkey brain in the period of early transient incapacity during gamma-irradiation at high dose rates].

Changes in the electroencephalogram (EEG) of Macaca fascicularis during early transient incapacitation (ETI) were shown to correlate with the dynamics of clinical manifestations of the damage. Irradiation caused desynchronization of EEG followed by a generalized retardation of its fluctuations reaching the maximum at the height of ETI. EEG disturbances in animals during the comatose phase of ETI indicated a severe inhibition of the brain cortex functional activity.

Animals↗
PubMedDate not supplied

[Total bioelectrical activity of the sensorimotor and visual cortices in rabbits during early neurological disorders from gamma irradiation at high doses].

The overall bioelectrical activity of the sensorimotor and visual brain cortex of rabbits was estimated during early neurological impairment caused by 120 Gy gamma-irradiation. The characteristic changes were revealed in the amplitude, form, energy spectrum and spatial biopotential synchronization. The changes in the bioelectrical activity of the brain were associated with the clinically displayed stages of the neurological process development.

Animals↗
PubMedDate not supplied

[Correlation between the complex of body reactions to exposure to acute hypoxia and individual radiosensitivity in irradiation at a dose of 200 Gr].

A study was made of a correlation between the response of basal metabolism to acute hypoxia and the life span of rats after irradiation resulting in the development of a cerebral form of radiation sickness. The more radiosensitive animals consumed a larger amount of oxygen, exhaled a lesser amount of carbon dioxide, and showed an increased normal expiratory exchange per minute. After the effect of acute hypoxia all the indices under study exhibited an opposite picture.

Acute Disease↗