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Biomedical subjects

D Lillystone

Publications and source records attributed to D Lillystone.

9 recordsLinked to original sources
PubMed1991

Renal disease presenting as severe unremitting colic.

We made a retrospective study of 1770 infants with colic. Of these infants 206 were classified as having severe colic. Six of the infants with severe colic failed to respond to intensive behaviour management and were found to have urinary infection. Later investigation revealed that all six had significant renal disease. We recommend that all infants with colic who do not respond rapidly to conventional management be investigated for urinary tract infections.

Colic↗
PubMed1984

Congenital dislocation of the hip. Incidence, and treatment of a local population group.

For a period of three months, the incidence of hip abnormality was determined in 450 consecutive deliveries at the Hornsby and Ku-ring-gai Hospital newborn nursery. Three children were found to have dislocatable hips, and received treatment with a von Rosen splint. In 60 babies, soft tissue clicks could be elicited but the joints were stable. These babies received no treatment, but were reassessed one and six weeks later. No hip abnormality was detected in any of them at the follow-up examination. A retrospective analysis of the records of the previous 450 newborn babies showed that nine had received treatment with double nappies; von Rosen splints had not been used. At the six-month follow-up examination, one of these nine babies had a dislocated hip. "Double nappies" as a treatment modality is still widely practised in Sydney, though neither the literature nor the results of our retrospective analysis support its efficacy.

Australia↗
PubMed1983

Ear anomalies, deafness and facial nerve palsy in infants of diabetic mothers.

Two children with an unusual combination of ear anomalies, deafness and facial nerve palsy are described. They are both children of insulin dependent diabetic mothers. There is no other family history of genetic deafness or congenital anomaly. The defects in both children may be accounted for by abnormal embryological development of the petrous temporal bone. It is postulated that poor metabolic control of the diabetes has led to these malformations.

Adult↗
PubMed1980

Pseudohypoaldosteronism.

10 infants are described with pseudohypoaldosteronism, 5 in detail and a further 5 briefly. They all presented with hyperkalaemia, urinary salt-wasting disease, and ostensibly normal renal and adrenocortical function. Diagnosis was established by demonstrating the greatly increased values of plasma renin activity and plasma aldosterone concentration, plus the increased excretion of aldosterone and its metabolites on gas chromatographic and mass spectrometric analyses of urine. The children were treated with sodium chloride supplements, up to 60 mmol/day, but by the time most of the infants were about a year old these could be stopped. Exogenous mineralocorticoids were without effect in those to whom they were administered. The precise aetiology of the condition remains conjectural; lack of renal tubular response to aldosterone seems probable. Pseudohypoaldosteronism may be more common than has been thought and new techniques for investigating salt-wasting disorders may show its true incidence.

Aldosterone↗