PubMed Health⌕ Search

Biomedical subjects

D R Seaman

Publications and source records attributed to D R Seaman.

10 recordsLinked to original sources
PubMed1999

Spinal pain syndromes: nociceptive, neuropathic, and psychologic mechanisms.

BACKGROUND: Pain continues to be the main symptom reported by patients. Frequently, clinicians incorrectly diagnose patients and resulting treatments are ineffective, which may promote the development of chronic pain. This situation may arise as a result of a lack of clarity in the literature regarding pain syndromes. OBJECTIVE: To discuss the differences between nociceptive, neuropathic, and psychologic induction of pain and provide important clinical correlates to aid in diagnosis and treatment. DATA SOURCES: The data were accumulated over a period of years by reviewing contemporary articles and books and subsequently retrieving relevant papers. Articles also were selected from MEDLINE searches and from manual library searches. DATA SYNTHESIS: Nociceptive pain syndromes are responsible for the majority of pain complaints in clinical practice. Care must be taken to avoid the common mistake of the diagnosis of neuropathic pain, which can lead to inappropriate treatments. CONCLUSION: Although the treatment of neuropathic pain is difficult, sufficient evidence in the literature demonstrates that the treatment of nociceptive pain should be multimodal and involve spinal manipulation, muscle lengthening/stretching, trigger point therapy, rehabilitation exercises, electrical modalities, a variety of nutritional factors, and mental/emotional support.

Acute Disease↗
PubMed1998

Dysafferentation: a novel term to describe the neuropathophysiological effects of joint complex dysfunction. A look at likely mechanisms of symptom generation.

BACKGROUND AND OBJECTIVES: Since the founding of the chiropractic profession, very few efforts have been made to thoroughly explain the mechanism(s) by which joint complex dysfunction generates symptoms. Save for a few papers, only vague and physiologically inconsistent descriptions have been offered. The purpose of this article is to propose a precise and physiologically sound mechanism by which symptoms may be generated by joint complex dysfunction. DATA SOURCES: The data was accumulated over a period of years by reviewing contemporary articles and books, and subsequently retrieving relevant papers. Articles were also selected from volumes 1-4 of the Chiropractic Research Archives Collection. The Nexus, published by the David D. Palmer Health Sciences Library, and In Touch, published by Logan College of Chiropractic Library, were reviewed and relevant articles were retrieved. Medline searches were found to be ineffective because appropriate key indexing terms were difficult to identify. DATA SYNTHESIS: The symptoms generated by joint complex dysfunction, such as pain, nausea and vertigo, are probably caused by increased nociceptive input and/or reduced mechanoreceptive input. CONCLUSIONS: Joint complex dysfunction should be included in the differential diagnosis of pain and visceral symptoms because joint complex dysfunction can often generate symptoms which are similar to those produced by true visceral disease.

Afferent Pathways↗
PubMedDate not supplied

The short golf backswing: effects on performance and spinal health implications.

BACKGROUND: Full recoil golf swings have been implicated in back pain and injury in golfers. Evidence suggests that a restricted backswing may reduce the potential for injury without compromising performance. OBJECTIVE: To examine both golf swing performance and selected muscular actions of the trunk and shoulder during a full recoil swing as compared with a modified short backswing. METHODS: Electromyographic (EMG) recordings were taken bilaterally from the lumbar, external oblique, latissimus dorsi, and right pectoral muscles in 7 golfers during a full recoil swing and a modified short backswing. High-speed videotape was used to measure back swing angle reduction. Clubhead velocity (CHV) and ball-contact accuracy were quantified by using a swing speed indicator and clubface contact tape, respectively. RESULTS: Shortening of the backswing by 46.5 degrees +/- 24.7 degrees had no effect on stroke accuracy as measured by mean deviation from the target spot on the club (19.0 +/- 7.8 mm vs 19.3 +/- 9.2 mm). CHV was not significantly reduced (33.9 +/- 2.5 m/s vs 31.2 +/- 2.2 m/s). However, EMG root-mean-square was decreased 19% in the right oblique muscle from 750 to 250 ms before impact (P < .05). During the acceleration phase, activation of left lumbar muscle decreased by 12%, whereas activation of right latissimus muscle increased by 21%. Although left lumbar muscle activity during the follow-through increased 14%, there was a substantial (17%) but nonsignificant decrease of activation of trunk muscles (P = .11). There was a general trend toward an increased activation of the shoulder musculature from 250 ms before impact to 500 ms after impact. CONCLUSION: These data support the idea that short backswings in golf may reduce trunk muscle activation and possibly reduce back injury and pain without negatively impacting swing accuracy or CHV. However, the short swing increases shoulder muscle activation and may, in turn, promote risk for shoulder injury.

Adult↗