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Biomedical subjects

D W Eggleston

Publications and source records attributed to D W Eggleston.

11 recordsLinked to original sources
PubMed2000

Improved laboratory results for fixed restorations using sequential tooth preparation.

Laboratory reference guides provided by the dentist help ensure optimum results in fixed prosthodontics. A technique is described in which a master cast is made before preparation of the last tooth to be treated. Through this technique, the laboratory prescription includes the dimensions of the new restorations in relation to those of the unprepared tooth replica on the master cast. Thus, the technician is afforded not only a precise guide to restoration contour and size, but also a helpful pretreatment record of the patient's vertical dimension.

Crowns↗
PubMed1989

A sign of the times.

Explore the source record for details and available documents.

Communicable Disease Control↗
PubMed1989

Dental amalgam: a review of the literature.

Since the 1800s, dental amalgam has been the most commonly used dental restorative material. Each year, dentistry in the United States uses over 100 tons of mercury, continuing a controversy regarding mercury's safety for patients and dental personnel.

Dental Amalgam↗
PubMed1987

Correlation of dental amalgam with mercury in brain tissue.

Data from this project demonstrate a positive correlation between the number of occlusal surfaces of dental amalgam and mercury levels in the brain (p less than .0025 in white matter). This is indirect evidence suggesting that mercury from dental amalgam fillings may contribute to the body burden of mercury in the brain. The toxic levels of mercury in human tissues have not been sufficiently investigated and the amount of mercury in human brain tissue from dental amalgam may or may not be clinically significant. Nevertheless, dental amalgam exposure should be considered in monitoring sources of mercury accumulation in human brain tissue.

Adolescent↗
PubMed1985

IgA nephropathy associated with dental nickel alloy sensitization.

We report a patient with documented IgA nephropathy in whom microscopic hematuria, proteinuria, and hypertension first occurred after placement of nickel alloy base dental crowns. Progressive proteinuria culminating in nephrotic-range proteinuria occurred parallel to increased nickel placement and dramatically resolved following nickel alloy removal. That immunologic alterations occur as a result of nickel exposure has already been suggested by the common occurrence of nickel contact dermatitis, often exacerbated by intraoral nickel placement, increased carcinogenesis in nickel refinery workers, and animal models of nickel-associated carcinogenesis. Our patient may represent an example of nickel-induced sensitization and associated IgA glomerulopathy. Further study of patients with immune-mediated glomerulopathy with attention to dental nickel exposure appears indicated.

Adult↗
PubMed1984

Effect of dental amalgam and nickel alloys on T-lymphocytes: preliminary report.

Preliminary data suggest that dental amalgam and dental nickel alloys can adversely affect the quantity of T-lymphocytes. Human T-lymphocytes can recognize specific antigens, execute effector functions, and regulate the type and intensity of virtually all cellular and humoral immune responses. Normal immune function depends on a proper quantity, quality, and ratio of T-lymphocyte helper and suppressor subsets. Further research may determine the frequency and magnitude of T-lymphocyte reduction and alteration by dental materials.

Adult↗
PubMed1980

The interrelationship of stress and degenerative diseases.

The following hypothesis has been proposed: Stressors--environmental and/or nutritional--can cause functional disorders. Functional disorders are abnormal neurology, endocrinology, and kinesiology without underlying organic pathology. The malfunction of these three systems is a major factor in the pathogenesis of systemic and dental degenerative diseases. Dental structural aberrations (malocclusion) can be perceived by the brain as major, 24-hour-a-day stressor agents, thus adding substantially to the total distress of the patient. In this manner, malocclusion becomes a significant factor in the pathogenesis of degenerative diseases. Under this hypothesis, malocclusion is the result of functional disorders, not genetics. Treatment of the entire patient cannot be overemphasized. The following suggestions can be made: 1. Conventional dental restorative treatment should be supplemented with nutritional therapy to eliminate nutrition as a distress agent. 2. Dental pathosis should be eliminated for optimum systemic health as well as preservation of the dentition.

Environment↗