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Biomedical subjects

E Besada

Publications and source records attributed to E Besada.

6 recordsLinked to original sources
PubMed2001

Hemodialysis vascular assessment by an ultrasound dilution method (transonic) in patients older than 65 years.

UNLABELLED: Malfunction of vascular access is one of the most frequent causes of morbidity and mortality in hemodialysis patients (HD). Early diagnosis makes possible the most frequent vascular access (VA) used in HD patients. The arteriovenous fistula (AVF), both autologous or heterologous, is the appropriate correction by an interventional radiologist or by surgery, before thrombosis appears. For this purpose, a follow-up of VA is mandatory. New technologies offer non-invasive methods for this purpose. In HD sessions ultrasound 'on line' and ultrasound-dilution techniques permit us to monitor vascular access in HD patients. Also transonic technology has been validated for this purpose, although the limitations of its use among elderly patients is unknown. Using the Transonic HD01 monitor, we studied vascular access in 45 patients in HD older than 65 years, and compared them with 47 patients who were younger than 65 years. The parameters analyzed were: effective flow Qt, recirculation, venous pressure and access flow. We found no significant differences between these parameters but in both groups found that the effective flow measure by Transonic was lower than that measured by a blood pump. Both groups contained patients who had no recirculation but had an access flow that was lower than expected. To rule out stenosis of VA in those patients, we performed an Eco Doppler confirming that all patients had stenosis. With this method, one can determines the access flow and thus predicts the possibility of future thrombosis. CONCLUSION: Our data confirm that one can evaluate VA in patients older than 65 years with Transonic HD01 monitor, and also in patients younger than 65 years. Due to the special characteristics of the vessels in elderly patients, Transonic HD01 monitor is a good method by which to monitor VA in them.

Aged↗
PubMed2001

Absent relative afferent pupillary defect in an asymptomatic case of lateral chiasmal syndrome from cerebral aneurysm.

INTRODUCTION: This report documents a case of an asymptomatic 34-year-old female who had right eye subtle temporal optic nerve pallor with absent clinically detectable relative afferent pupillary defect. CASE REPORT: This presentation was secondary to a fusiform paraclinoid aneurysm resulting in lateral chiasmal syndrome. Visual fields demonstrated a nasal depression OD. Neuroimaging studies revealed the presence of a fusiform paraclinoid aneurysm compressing the right optic nerve and optic chiasm. The lesion was treated surgically without additional visual loss nor systemic and neurological complications. CONCLUSIONS: A clinically detectable relative afferent pupillary defect may be absent in unilateral optic nerve pallor and/or visual field loss and does not preclude the need to attempt to identify an underlying etiology. Neuroradiological examination of orbital and cranial structures is an imperative protocol in the appropriate management of this potential life- and vision-threatening condition.

Adult↗
PubMed2000

Post-streptococcal uveitis.

BACKGROUND: We report additional evidence supporting the association between the occurrence of bilateral nongranulomatous uveitis and previous infection by group A streptococci--i.e., post-streptococcal syndrome. METHODS: As shown through a series of case reports, physical examination and laboratory blood analysis--including antistreptolysin O (ASO) antibody titer--were ordered due to a recent history of sore throat and presence of nongranulomatous bilateral anterior uveitis. RESULTS: Serological laboratory testing revealed elevated ASO antibody titer. The etiology of the uveitis was attributed to streptococcal infection. Bilateral uveitis responded to topical corticosteroids. Systemic antibiotic treatment was used to treat possible post-streptococcal syndrome sequelae with resolution of symptoms. CONCLUSION: Uveitis should be considered a possible manifestation of post-streptococcal syndrome. ASO antibody quantification should be included in the serologic testing performed in evaluating the cause of seemingly idiopathic bilateral nongranulomatous anterior uveitis associated with signs and symptoms that suggest previous streptococcal infection.

Adolescent↗
PubMed1996

Hypothyroidism--a possible etiology of open-angle glaucoma.

BACKGROUND: Recent studies have shown an association between hypothyroidism and primary open-angle glaucoma (POAG). When hypothyroidism occurs, accumulation of hyaluronic acid within the trabecular meshwork may cause an increase in outflow resistance and intraocular pressure (IOP). This increase may be reversible with use of systemic treatment. METHODS: A hypothyroid patient is described whose IOP inversely varied with the use or the discontinuation of systemic hypothyroid medications. RESULTS: The patient's IOP was controlled when she complied with systemic treatment. CONCLUSIONS: In some cases, hypothyroidism may contribute to the genesis of open-angle glaucoma. Systemic treatment alone may be adequate to prevent progression of a glaucomatous diseased state. It may be prudent to investigate the presence of undiagnosed hypothyroidism in individuals with poorly controlled POAG, in individuals with normal-tension glaucoma, and in newly diagnosed cases.

Adult↗
PubMed1994

Clinical diagnosis of recurrent herpes simplex blepharitis in an adult: a case report.

BACKGROUND: Herpes simplex (HSV) blepharitis occurs usually in children as a primary infection. Though primary HSV blepharitis is also observed in adults, recurrent HSV blepharitis involving the same area of the eyelid is unusual in this group. Two clinical types have been identified, one consisting of vesicular type lesions and a second characterized by erosive-ulcerative insults. Presented in this article is a case of an adult with herpes simplex blepharitis (presumed recurrent at the same generalized eyelid region), manifesting erosive-ulcerative lesions. Review of this ocular condition, treatment modalities, and hypothetical modes of transmission is given.

Adult↗