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Biomedical subjects

E Höring

Publications and source records attributed to E Höring.

24 records · Page 2Linked to original sources
PubMed1989

[Severe cholestasis in congenital choledochus cyst].

A nineteen year old female was admitted to the hospital presenting with sudden onset of severe jaundice and the biochemical constellation of cholestasis. The sonographically and radiologically diagnosis of choledochal cyst was confirmed intraoperatively. The choledochal cyst of type IVa reached from the papilla until the hepatocholedochal junction. A lateroterminal choledochojejunostomy with a Y-Roux-anastomosis was constructed. The patient became symptom-free after the operation.

Adult↗
PubMed1988

[Portal hypertension due to thromboses of the portal, splenic and mesenteric veins in congenital protein c deficiency].

In a 39-year-old man there occurred, over a period of four years, mesenteric vein thrombosis with haemorrhagic infarction of the intestines, as well as multiple bleedings from oesophageal varices due to portal hypertension caused by portal vein thrombosis. Search for the cause of the thrombosis tendency revealed congenital protein C deficiency. Splenectomy and a disconnection operation were performed, followed by administration of anticoagulants. Since then the patient has been free of symptoms, and there have been no further variceal bleedings or thromboses.

Adult↗
PubMedDate not supplied

Influence of ascites on the chemotaxis of granulocytes in patients with cirrhosis.

Spontaneous bacterial peritonitis is a specific infectious complication in liver cirrhosis. The reasons for the preferred location of infection on the peritoneum are not clear. The aims of the present study were to ascertain whether hepatogenic ascites fluid is chemotactically effective, what part is played by complement factor C3 and whether there are inhibitors of chemotaxis in ascites. Chemotaxis of granulocytes in serum and ascites fluid was measured in 18 patients with cirrhosis and ascites and in 18 healthy individuals using the Boyden chamber method. In the patients, the chemotactic effect of serum was reduced significantly. Ascites fluid had lower chemotactic activity than autologous serum (P < 0.01), directly correlated to C3 levels (P < 0.025). There was a significant correlation between chemotaxis in serum and in ascites fluid (P < 0.005). Adding ascites fluid to serum led to reduction of chemotactic activity only in the patients (P < 0.025). In conclusion, the chemotactic effect of ascites fluid is considerably lower than that of serum and is proportional to local concentrations of C3. Chemotaxis-inhibiting factors can also be identified in ascites fluid, their pathogenetic relevance being limited.

Adult↗