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Biomedical subjects

E Junco

Publications and source records attributed to E Junco.

14 recordsLinked to original sources
PubMed1991

[Membranous nephritis after renal transplantation].

8 cases of membranous glomerulonephritis (MG) after renal transplants (RT) are presented; one being a recurrence of the original disease and the other 7 due to a different cause of renal insufficiency. The total incidence of MG after transplantation was 1.63%; 1.39% being the incidence of MG of new cases. Only 1 patient showed decrease of renal function and in this case the MG was accompanied by chronic rejection lesions. There was no sign of neoplasias nor drugs producing MG. As far as chronic infections are concerned, only one patient showed B antigen and it was not observed during the immunofluorescent test in the biopsy. 6 patients had urological complications after the renal transplant (3 cases of urinary fistula; 2 cases of obstructive uropathy; 1 case of short ureter). 2 patients experienced the start of hemodialysis due to focal and segmentary glomerulosclerosis. The beginning of proteinuria commences between 2 and 23 months after the RT (median 13,0 +/- 7,5 moths); with a range of between 2.0 and 12.0 gr/day (median: 6.8 +/- 3,2 Z gr/day), this being nephrotic in 4 cases. Proteinuria improved 1 case, and persisted in the other patients at the same level registered previous to the diagnosis. MG is a non-frequent complication or RT and is usually benign. Patients with post-transplant urologic complications could be considered to have a higher risk of developing a MG "de novo".

Adolescent↗
PubMed1979

[Therapeutic guides in hypertonic dehydration (author's transl)].

Physiopathology of hypertonic dehydration is revised. The bases of treatment are discussed; they are based on: 1. Basal requirements of fluids. 2. Slow correction of deficit. 3. Replacement of maintaining losses. Total fluids support will carry to rehydration in 2 or 3 days. In the most severe cases peritoneal dialysis is associated. The indications, technique and realization of peritoneal dialysis during hypertonic dehydration are discussed.

Dehydration↗
PubMed1979

[Treatment and evolution of shock and acidosis in hypertonic dehydration (author's transl)].

Authors analyse treatment and clinical and analytical evolution of 80 patients with hypertonic dehydration, depending on shock and acidosis. Several considerations concerning treatment and its' influence on evolution of different patients, emphasizing both therapeutic methods: with or without use of peritoneal dialysis, according to the values of initial natraemia, are made.

Acidosis↗
PubMed1979

[Metabolic evolution of hypertonic dehydration treated with fluidotherapy: revision of 40 infants (author's transl)].

Authors study the metabolic evolution of osmolarity, natraemia, chloremia, kaliemia and uremia of 40 infants with hypertonic dehydration. They divide the serie into two groups in accordance with initial value of natraemia: more or less than 170 mEq./l. The slow average decreases by hour of osmolarity, natraemia and chloremia were evaluated, and that justifies good evolution of hypertonic dehydration and minimal neurological repercussion found. Renal function in this metabolic condition is discussed. The usefulness of the fluidotherapic technique employed is demonstrated.

Chlorides↗
PubMed1979

[Metabolic evolution of hypertonic dehydration with combined treatment of fluidotherapy and peritoneal dialysis: revision of 22 infants (author's transl)].

Metabolic evolution of osmolarity, natraemia, chloremia, kaliemia and uremia of 22 infants with severe hypertonic dehydration, treated with fluidotherapy and peritoneal dialysis, is revised. Peritoneal dialysis was prepared in accordance with a special method for this metabolic condition. The serie was divided into two groups, according to initial value of natraemia: more or less than 170 mEq./l. Average decreases per hour of osmolarity, natraemia and chloremia were evaluated, as well as the influence of peritoneal dialysis on body fluids composition. Indication of peritoneal dialysis specially prepared for severe cases is discussed.

Chlorides↗
PubMed1979

[Contribution to acid-base metabolism in hypertonic dehydration by the study of "fraction R" (underterminate anions) (author's transl)].

A study on the value of "R fraction" of plasma electrolites (undetermined anions), in order to know better the metabolic acidosis of the hypertonic dehydration is achieved. "R fraction" value was obtained by the formulae: R fraction = plasma N+ --(plasma Cl- + plasma HCO3-). The mean values in patients with hypertonic dehydration was: 12.4 +/- 8.3 mEq./l., more than the normal standard data of our laboratory: 8.55 +/- 3.4 mEq./l. Authors make two groups in accordance with the "R" value: one with low "R fraction" (25 cases): 4.79 +/- 4.70 mEq./l.; and another with high "R fraction" (32 cases): 20.04 +/- 6.6 mEq./l., and compare the differences (ethiologic, clinic, shock and metabolic) between the two groups. It is confirmed that acidosis in hypertonic dehydration is essentially hyperchloremic. It was speculated on the "anion gap" and the total value of chloride in this dehydration.

Acid-Base Equilibrium↗
PubMed1978

[Adrenal function in topic corticotherapy].

It has been shown that corticosteroids for dermatological use produce an inhibition of the adrenal function, and it is considered that in our milieu these products are used without any precise indication. In this study, 29 ambulatory children from 3 to 15 years of age were selected, who were suffering from dermatoses with different degrees of extension, on whom urinary 17-ketosteroids, 17-ketogenicsteroids and serum cortisol were measured before and after non-occlusive treatment of two weeks, using 0.01 percent fluorocorticoids and controls after 24 hours and 8 days. A significant drop of the 17-keratogenicsteroids was found in patients studied at the end of the treatment, with immediate recovery. Additionally it was observed, in patients who had over 30 percent of skin area involved, that there was a decrease in serum cortisol, which persisted even 24 hours after discontinuing treatment, with normal levels in a measurement taken eight days later. It is therefore concluded that glucocorticoids applied topically are capable of inhibiting adrenal function.

17-Ketosteroids↗
PubMed1977

[18 short arm deletion. Report of one case (author's transl)].

A two month old boy with multiple malformations: mental retardation, microcephaly, hyperterloism, displasic ears, hypospadias, unilateral cryptorchidism and holoprosencephaly is presented. In leukocytes culture, patient shows a deletion of the short arm of a 18 chromosome. This aberration apears "de novo" in this patient.

Abnormalities, Multiple↗
PubMedDate not supplied

HbA1, height velocity and weight gain as indicators of metabolic control in type I diabetic children. A 5 year survey.

The purpose of this study was to evaluate the metabolic control of the type I diabetic patients seen from 1984 to 1988. We analyzed the results of HbA1, height velocity and weight gain. Results showed that from 1984 1985 a 90% of diabetics were in poor metabolic control with HbA1 > 11% (good control < 11%). For 1988 the percentage of poor controlled patients descended to 78%. On the other hand, these patients observed a significant decrease in height velocity and weight gain compared with diabetics with HbA1 < 11% (p < 0.005 and p < 0.001). In addition, diabetics in poor metabolic control were under 10 and 3 number percentiles of the weight and growth diagrams. Finally, we found a significant correlation between HbA1 and height velocity as well as HbA1 and weight gain (r = -0.77 and r = -0.79; p < 0.001 for both). Our results showed that a great percent of our patients were in poor metabolic control with a decreased height velocity and weight gain.

Adolescent↗
PubMedDate not supplied

[Budd-Chiari syndrome in childhood by hipoplasia of suprahepatic veins. Clinical and pathological study of a case (author's transl)].

A case of Budd-Chiari syndrome with hypoplasia of suprahepatic veins is reported. A bibliographic review is made. Etiologic and anatomic factors are analyzed. Its clinical, analytic, pathological and embryologic findings are compared with those of other authors. The rarity of this case, for the age of presentation as much as for its etiology is pointed out.

Arteriovenous Malformations↗