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Biomedical subjects

E M Ahrens

Publications and source records attributed to E M Ahrens.

5 recordsLinked to original sources
PubMed1989

Cutaneous reactions to drugs used for rheumatologic disorders.

Cutaneous reactions to medications probably represent the most common manifestation of drug reactions. The diversity of cutaneous eruptions produced by drugs provide a challenge in searching for the mechanisms producing the reaction. Many eruptions are due to a form of allergic hypersensitivity, while others may be idiosyncratic, due to a metabolic abnormality, or represent a cumulative phenomenon. This article discusses the diagnosis of drug-induced cutaneous reactions by reviewing specific drugs commonly used in rheumatologic therapy.

Acetates↗
PubMed1988

Granulomatous cutaneous rheumatoid vasculitis.

Rheumatoid vasculitis is a term that includes many vascular reactions from capillaritis to severe systemic necrotizing vasculitis occurring in a patient with rheumatoid arthritis (RA), but generally has been used to describe an immune complex reaction. The presence of vasculitis in the patient with RA has often been associated with a poorer prognosis; however, the prognosis is dependent on the presence of systemic disease. We report a patient with RA in whom multiple purpuric, cutaneous nodules, diffuse interstitial fibrosis, and high levels of circulating immune complexes developed. The cutaneous disease was characterized by a granuloma formation secondary to leukocytoclastic vasculitis.

Aged↗
PubMed1987

Cutaneous reactions to neurologic drugs.

Neurologic drugs are often implicated as the causative agents producing skin eruptions. In our highly medicated society, correctly identifying the offending medication is often difficult. Determining the causative agent involves knowledge of the common offenders, their frequency of occurrence, and understanding the commonly produced reaction patterns in the skin.

Drug Eruptions↗
PubMed1986

Dapsone-induced peripheral neuropathy.

A young man with dermatitis herpetiformis developed fatigue and neurologic complaints 4 years after he began oral dapsone therapy. Neurologic examination and nerve conduction studies confirmed the presence of a combined motor and sensory peripheral neuropathy. The symptomatic improvement reported by the patient was supported by improvement in the nerve conduction studies after cessation of dapsone therapy. Substitution of sulfapyridine did not adversely affect the resolution of his neuropathy.

Administration, Oral↗