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Biomedical subjects

E M Hetherington

Publications and source records attributed to E M Hetherington.

At least 19 recordsLinked to original sources
PubMed2001

Differential parenting as a within-family variable.

Nonshared environmental influences have been found to be important for adolescent development. This study of 516 families investigated whether differential parental negativity or warmth is linked to adolescent adjustment apart from the effect of the level of parenting toward each child separately. After accounting for level of parental treatment to the adolescent, the authors found that differential parenting to the siblings contributed unique variance in adjustment. Significant interactions were found between level of parenting and differential parenting. In each case, differential parenting was more strongly linked to adjustment when the level of parenting was low in warmth or high in negativity. These results are indirect evidence that differential parenting can be considered a within-family influence on sibling adjustment and as direct evidence that nonshared environmental factors may systematically vary in strength between families.

Adaptation, Psychological↗
PubMed2000

Contemporary research on parenting. The case for nature and nurture.

Current findings on parental influences provide more sophisticated and less deterministic explanations than did earlier theory and research on parenting. Contemporary research approaches include (a) behavior-genetic designs, augmented with direct measures of potential environmental influences; (b) studies distinguishing among children with different genetically influenced predispositions in terms of their responses to different environmental conditions; (c) experimental and quasi-experimental studies of change in children's behavior as a result of their exposure to parents' behavior, after controlling for children's initial characteristics; and (d) research on interactions between parenting and nonfamilial environmental influences and contexts, illustrating contemporary concern with influences beyond the parent-child dyad. These approaches indicate that parental influences on child development are neither as unambiguous as earlier researchers suggested nor as insubstantial as current critics claim.

Animals↗
PubMed2000

Relationship between perceptual differences of parenting and adolescent antisocial behavior and depressive symptoms.

The relation between adolescent negative adjustment and differences in parent-child ratings of parents' warmth and negativity was examined with a national sample of 720 families. It was predicted that perceptual differences (PDs) would be linked to more negative adjustment. Adjustment was regressed on PDs, which were calculated as absolute differences between parent and child ratings of parenting. Results showed that PDs were significantly associated with adjustment independent of the level of parenting behavior. Associations differed by gender for PDs over maternal verbal aggression. Some of the most important results were curvilinear effects indicating that both high and low, but not medium, levels of PDs are linked with maladjustment. Finally, differences between younger and older adolescents were found: The linear relationship between PDs over parental negativity and maladjustment disappeared for older adolescents.

Adolescent↗
PubMed1999

Relationships between parenting and adolescent adjustment over time: genetic and environmental contributions.

The predictive association between parenting and adolescent adjustment has been assumed to be environmental; however, genetic and environmental contributions have not been examined. This article represents one effort to examine these associations in which a genetically informative design was used. Participants were 395 families with adolescent siblings who participated in the Nonshared Environment in Adolescent Development (D. Reiss et al., 1994) project at 2 times of assessment, 3 years apart. There were 5 sibling types in 2 types of families: 63 identical twins, 75 fraternal twins, and 58 full siblings in nondivorced families and 95 full, 60 half, and 44 genetically unrelated siblings in stepfamilies. Results indicate that the cross-lagged associations between parental conflict-negativity and adolescent antisocial behavior and depressive symptoms can be explained primarily by genetic factors. These findings emphasize the need to recognize and examine the impact that adolescents have on parenting and the contribution of genetic factors to developmental change.

Adolescent↗
PubMed1999

Marital interaction in dyadic and triadic contexts: continuities and discontinuities.

While the systemic metaphor used in much current family research requires examination of the interrelationships among individuals, relationships, and the family as a whole, work on triadic relationships has generally been missing. This research examined the presence of second-order effects in marital interaction: changes in interactions between spouses when the husband-wife dyad became a parent-parent-child triad. Results indicated the presence of consistent context effects. Parental behavior when alone was not a good predictor of parental behavior in the presence of a child: behaviors occurred at significantly lower levels in parental dyads than in parent-parent-child triads, and correlations across the two contexts were less than consistent. Results are discussed in light of their implications for observations of families.

Adult↗
PubMed1999

The adjustment of children with divorced parents: a risk and resiliency perspective.

This review addresses major questions about divorce, around which much contemporary research is oriented. These involve questions of the consequences of divorce for the adjustment of children and the vulnerability and resiliency of children in coping with divorce, whether children are better off in a conflictual intact family situation or a divorced family, and how mothers, fathers, and clinical or educational interventions can moderate the effects of divorce. Although research in the past decade has yielded considerable information about these questions, issues that need further investigation are also presented.

Adaptation, Psychological↗
PubMed1999

Adolescents' relationships to siblings and mothers: a multivariate genetic analysis.

Research has consistently demonstrated that children's behavior toward their siblings tends to resemble interactions occurring in the parent-child relationship. This study examined the relative contributions of genetic and environmental influences to the covariation between sibling relationships and mother-adolescent relationships. Reported and observed family interactions were assessed for 719 same-sex sibling pairs of varying degrees of genetic relatedness. The covariance between mother-adolescent and sibling interactions was decomposed into genetic, shared, and nonshared environmental components. The overlapping effects of shared environment on the two relationship subsystems explained most of the covariance. Smaller but significant genetic and nonshared environmental effects were also found. The consistency of these findings with family processes, such as modeling, is discussed.

Adolescent↗
PubMed1999

The importance of shared environmental influences in explaining the overlap between mother's parenting and sibling relationships: reply to Neale (1999)

This article addresses concerns raised by M. C. Neale (1999) in his commentary on the D. A. Bussell et al. (1999) Nonshared Environment in Adolescent Development (NEAD) study. These concerns fall into two categories: (a) model assumptions and sample design and (b) testing of alternative models. The validity of the assumptions of quantitative genetic models is a concern for all researchers in this area. Discussion of those assumptions in this reply is brief and focuses on those most relevant to the NEAD sample. The two alternative models proposed by Neale were designed to provide alternatives to the large shared environmental effect found in the original report of Bussell et al. Because these alternative models did not provide a better fit, the appropriateness of Bussell et al.'s basic model and the importance of shared environmental influences for explaining the association among family subsystems are supported.

Adolescent↗
PubMed1998

Family systems and adolescent development: shared and nonshared risk and protective factors in nondivorced and remarried families.

The primary goal of this research is to increase the goodness-of-fit between the theoretical tenets of family systems theory and quantitative methods used to test systems hypotheses. A family systems perspective is applied to two specific research questions concerning family influences on adolescent development: To what extent are familial risk and protective factors for psychopathology and competence shared or not shared by siblings and are different family relationship patterns associated with optimal adolescent adjustment in nondivorced and remarried families? Multirater and multimethod data from a national sample of 516 nondivorced and remarried families from the Nonshared Environment and Adolescent Development (NEAD) project were examined using a combination of cluster, factor, and regression analyses. Results indicated that the effects of an individual relationship on adolescent adjustment is moderated by the larger network of relationships in which it is embedded. Evidence for nonshared familial processes in predicting adolescent psychopathology was also found but only in a subset of families, and the mechanisms of influence were neither main effects nor linear, as has been assumed by research to date. Results are discussed in light of family systems models of relationship influence on development. These results illustrate how family systems theory provides a specific example of contextualism as regards the development of psychopathology in adolescence.

Adaptation, Psychological↗
PubMed1998

What matters? What does not? Five perspectives on the association between marital transitions and children's adjustment.

This article presents an analysis of 5 views of factors that contribute to the adjustment of children in divorced families or stepfamilies. These perspectives are those that emphasize (a) individual vulnerability and risk; (b) family composition; (c) stress, including socioeconomic disadvantage; (d) parental distress; and (e) disrupted family process. It is concluded that all of these factors contribute to children's adjustment in divorced and remarried families and that a transactional model examining multiple trajectories of interacting risk and protective factors is the most fruitful in predicting the well-being of children.

Adaptation, Psychological↗
PubMed1998

Adolescent perceptions as mediators of parenting: genetic and environmental contributions.

Explaining how genetic factors contribute to associations between parenting and adolescent adjustment is an important next step in developmental research. This study examined the mediating effect of adolescent perceptions on these associations and the genetic and environmental influences underpinning the mediated relationship. Parent, adolescent, and observer ratings of parenting and adolescent adjustment were used in a genetically informative sample of 720 same-sex sibling pairs from 10 to 18 years old. Adolescent perceptions of parenting did significantly mediate a composite measure of parental conflict-negativity and adolescent antisocial behavior and depressive symptoms. The most substantial genetic contributions to the association between parenting and adolescent maladjustment were those mediated by adolescent perceptions. Once genetic and environmental contributions to adolescent perceptions of parenting were removed, shared environmental factors became more important for the remaining direct association.

Adolescent↗
PubMed1998

Co-occurrence of depressive symptoms and antisocial behavior in adolescence: a common genetic liability.

Recent reviews of research on child and adolescent psychopathology have highlighted the consistently high rates of co-occurring dimensions of psychopathology, particularly between internalizing and externalizing disorders, and have suggested that further research examining the causes of co-occurring syndromes is needed. The authors examined this question in a national sample of 720 same-sex adolescent siblings between 10 and 18 years of age consisting of monozygotic and dizygotic twins, full siblings, half siblings, and unrelated siblings. Composite measures of adolescent and parent reports and observational measures of depressive symptoms and antisocial behavior were subjected to behavioral genetic models that examine the genetic and environmental influences on individual differences in each dimension as well as in the co-occurrence between dimensions. Results indicated that approximately half of the variability in depressive symptoms and antisocial behavior is attributed to genetic factors; shared and nonshared environmental influences were also significant. The co-occurrence of depressive and antisocial symptoms was explained by genetic and shared and nonshared environmental influences. Specifically, approximately 45% of the observed covariation between depressive and antisocial symptoms could be explained by a common genetic liability. Results are interpreted in light of contribution of genetic studies to debates on child and adolescent psychopathology.

Adolescent↗
PubMed1998

Genetic contributions to continuity, change, and co-occurrence of antisocial and depressive symptoms in adolescence.

In adolescence, antisocial and depressive symptoms are moderately stable and modestly correlated with each other. We examined the genetic and environmental origins of the stability and change of antisocial and depressive symptoms and their co-occurrence cross-sectionally and longitudinally in a national sample of 405 adolescents. Monozygotic (MZ) and dizygotic (DZ) twins and full, half, and unrelated siblings 10-18 years of age from non-divorced and step-families were studied over a 3-year period. Composite measures of adolescent self-reports, parent reports, and observational measures of antisocial and depressive symptoms were analysed in multivariate behavioural genetic models. Results indicated that the majority of the stability in and co-occurrence between dimensions could be accounted for by genetic factors. Non-shared environmental risks and, for antisocial symptoms, shared environmental risks also contributed to the stability. Genetic influences on change were observed, but only for antisocial behaviour. In addition, the longitudinal association between antisocial behavioural and later depressive symptoms was also found to be genetically mediated, but this effect was nonsignificant after controlling for stability. Results were discussed in light of the potential contributions of development behavioural genetic research in understanding individual differences in the stability and change of maladjustment.

Adolescent↗
PubMed1997

Dimensions and disorders of adolescent adjustment: a quantitative genetic analysis of unselected samples and selected extremes.

One of the fundamental questions for developmental psychopathology concerns the etiological links between the normal and abnormal. To what extent do disorders differ quantitatively or qualitatively from variation in the normal range? Genetic research on the normal and the abnormal differs in terms of concepts, methods, statistics, and target audiences. An approach, referred to as "DF" analysis, provides a framework for integrating these two worlds of genetic analysis. We applied traditional correlational analyses as well as DF (DeFries & Fulker, 1985) analyses to mother and father ratings of adjustment of adolescent siblings in a 3-year longitudinal twin and step-family study. At wave 1, the sample included 720 sibling pairs (average age of 12.9 years for the younger sibling and 14.5 years for the older siblings) and, in wave 2, 395 pairs still living at home. Both correlational analyses of the entire sample and DF analyses of selected extremes suggested moderate genetic influence and modest shared environmental influence for internalizing and externalizing behavior problems. Similar estimates were found for unselected individual differences and selected extreme groups. A framework is proposed that focuses on quantifying the etiologies of disorders (QED) as measured on continuous dimensions.

Adjustment Disorders↗
PubMed1996

Using MZ differences in the search for nonshared environmental effects.

Behavioural genetic research has shown that environmental influences relevant to the development of individual differences are largely nonshared in origin. That is, environmental influences do not make children in the same family similar to one another, however, little is known about which specific aspects of the environment are responsible. Identical (MZ) twins provide a uniquely powerful tool to search for specific nonshared environmental influences independent of nonshared genetics because such twins do not differ genetically. The sample consisted of 93 MZ twin pairs aged 10-18 years from the Nonshared Environment and Adolescent Development (NEAD) project. Reports of parental negativity and adolescent adjustment were obtained from parents, ratings of videotaped observations, and the adolescents themselves. Relative differences of mothers' and fathers' negativity within MZ pairs correlated significantly with MZ differences in antisocial behaviour. The correlations were moderate for within-reporter associations, but were negligible for associations between reporters. Possible interpretations for these source-specific findings are discussed.

Adolescent↗