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E Orinius

Publications and source records attributed to E Orinius.

15 recordsLinked to original sources
PubMed1989

Exudative pericarditis in sarcoidosis. A case report and echocardiographic study.

Involvement of the pericardium in sarcoidosis is infrequent as earlier reported. The involvement may be accompanied by pericardial effusion. We report the case of a 43 year-old man with non-obstructive hypertrophic cardiomyopathy for almost two decades who developed an effusion in the pericardial sac. As pericardial effusion is not a part of this disease and no other common cause was found we believe that the exudation was caused by sarcoidosis which started one year earlier. This finding initiated an echocardiographic study in 18 consecutive sarcoid patients, 8 with acute and 10 with chronic disease. None had pericardial effusion and only one demonstrated a thickening of the pericardial tissue.

Adult↗
PubMed1987

The Björk-Shiley 70 degree convexo-concave prosthesis strut fracture problem (present state of information).

Between June 1980 and June 1983 4028 Björk-Shiley 70 degree convexo-concave prosthetic heart valves were distributed and implanted in Australia, Canada, Europe and South Africa. As of March 1986, a total of 52 outlet strut fractures (1.29%; 70% CL: 1.1%-1.5%) have been reported from 29 implant institutions in 12 countries. The majority (82.7%) occurred in Europe. Intervals between implantation and fracture were 13 days to 45.3 months (mean: 18.4 months; 70% CL: 16.6 months-20.1 months). The mortality rate after strut fracture was 78.7% (70% CL: 72.5%-84.9%). Upon stratification of the fracture by valve sizes and types it becomes evident that 75% (70% CL: 68.8%-81.2%) of all fractures are related to the sizes 29 mm to 33 mm (which virtually represent the same valve size) and predominantly to mitral valves (p less than 0.01). The large valves again have been stratified into two subsets, namely those fabricated from flanges originally machined as Björk-Shiley 60 degree convexo-concave valves (group I) and later produced valves machined initially to 70 degree specifications (group II). In group I the fracture rate was 5.2% (70% CL: 4.2%-6.2%) versus 1.6% (70% CL: 1.1%-2.1%) in group II (p less than 0.01), which identifies the group I 29 mm-33 mm Björk-Shiley 70 degree convexo-concave valves as the highest risk group for strut fracture. The rates are based upon all available information as of March 16, 1986.(ABSTRACT TRUNCATED AT 250 WORDS)

Heart Valve Prosthesis↗
PubMed1979

Prognosis in hypertrophic obstructive cardiomyopathy.

Thirty-eight non-operated patients with hypertrophic obstructive cardiomyopathy (HOCM) were followed for 1--18 years (mean 8). Twelve patients died, nine of them instantaneously without any other apparent cause of death. The symptoms on the first admission did not discriminate between those who died and those who survived during the observation period, nor did the findings at heart catheterization at rest or left ventricular angiocardiography. However, cardiac enlargement on the first chest X-ray was significantly more common in the decreased group, 75% against 27%, as was the complete absence of a q-wave in lead III on the first ECG, 83% against 42%. The combination of complete lack of q in lead III and cardiac enlargement on chest X-ray at the initial examination was present in 9 of the 12 deceased (75%) and in only 3 of 24 survivors (13%). This can be used to select patients for long-term prophylaxis against ventricular fibrillation which, according to the literature, is the main mechanism of instantaneous death in HOCM.

Adolescent↗
PubMed1978

The temperature course in acute myocardial infarction.

The rectal temperature course was studied retrospectively in 192 consecutive patients with acute myocardial infarction (AMI). The ordinary temperature course after AMI was characterized by four points: 1. The morning temperature on the first day in hospital was seldom above 38.2 degrees C. (in four of 50 cases) 2. The maximal morning temperature was seldom recorded before day two or after day five in hospital (in five of 150 cases) 3. The maximal morning temperature seldom reached above 39.0 degrees C. (in one of 150 cases) 4. The morning temperature seldom increased more than 0.6 degrees C. after day five in one or more steps (in nine of 150 cases). Seventy-four per cent of the patients were afebrile after one week, and 96% after two weeks. Patients with higher SGOT (ASAT) maxima had longer duration of fever. Eleven per cent of the patients did not have fever at all.

Body Temperature↗
PubMed1978

Supraventricular tachyarrhythmias in acute myocardial infarction.

Onset of atrial tachycardia, flutter or fibrillation occurred in 11% of 274 consecutive patients with acute myocardial infarction (AMI). Atrial tachycardia started about 24 hours and atrial flutter/fibrillation about 72 hours after onset of AMI symptoms. Left heart failure, diagnosed as pulmonary rales or frank pulmonary edema, was not more common in these patients before onset of tachyarrhythmias than among the rest of the patients. On the other hand, a notching of the P wave in lead CR-was significantly more common in the patients with atrial fibrillation (67%). In most of these cases the terminal P force in lead CR1 was not negative as in so-called left atrial enlargement. These findings suggest that atrial conduction disturbances might be a basis of atrial fibrillation in AMI.

Acute Disease↗
PubMed1977

Restricted lignocaine prophylaxis in acute myocardial infarction.

In a consecutive series of 274 AMI cases ventricular tachycardia (VT), defined as three or more ventricular premature beats (VPBs) in succession but not VPBs, has been used as the indication for ventricular fibrillation (VF) prophylaxis. No primary VF occurred, and this fits with the hypothesis of VT as a sufficient indication for prophylaxis against primary VF. Six patients developed complicating VF (preceded by rales or hypotension but not frank pulmonary edema or shock). Four of the six patients (67%) had VT (0-1.5 hours) before VF, while the mean VT incidence of the six corresponding monitoring periods in 247 non-VF patients was 5%. Three of the four VT patients were on lignocaine/procainamide when VF developed. Thus, VT is acceptable as the only warning arrhythmia even in complicating VF but antiarrhythmic drugs do not seem to have the same prophylactic efficacy in complicating VF as in primary VF. Another 21 patients developed VF during shock, frank pulmonary edema or manipulating a pacemaker catheter within the heart.

Acute Disease↗
PubMed1975

Diazepam in cardioversion.

Diazepam has been used to an increasing extent in cardioversion, since avoiding general anaesthesia simplifier the procedure. The present study concerns the effect of diazepam on BP and blood gases in 13 cases of cardioversion. A moderate fall of both systolic and diastolic BP occurred. The arterial pO2 and pCO2 did not change significantly.

Aged↗
PubMed1975

Creatine phosphokinase after submaximal physical exercise in untrained individuals.

Serial estimations of total serum creatine phosphokinase (CPK) have been performed before and during 18-49 hours after submaximal physical exercise in 17 untrained individuals, mean age 50 years. The maximal CPK increase after exercise was 32 mU/ml (73%). The serum CPK did not exceed the upper normal limit (130 mU/ml) except in one individual (150 mU/ml). The maximal CPK increase in patients with acute myocardial infarction (AMI) varied between 101 mU/ml (133%) and 2 260mU/ml(3 790%), mean 900 mU/ml (1 184%). As the maximal CPK elevation in AMI occurs within the same period, it seems that heavy physical work of short duration just before the onset of symptoms will very seldom impair the diagnosis of AMI with the CPK technique used.

Adult↗
PubMed1975

Creatine phosphokinase following cardioversion.

Serial estimations of total serum creatine phosphokinase (CPK) have been performed before and during 18-51 hours after cardioversion of supraventricular tachyarrhythmias in 12 patients without acute myocardial infarction (AMI). The maximal CPK rise was 78 mU/ml (110%) and the CPK did not exceed the upper normal limit (130 mU/ml) in more than two patients (149 respectively 156 mU/ml). The CPK rise we have observed in a series of AMI patients varied between 101 (133%) and 2 260 mU/ml (3 780%), mean 900 mU/ml (1 184%). Therefore, cardioversion performed as described seldom seems to interfere with diagnosing AMI by serial estimations of serum CPK during the next 24 hours.

Aged↗