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Biomedical subjects

E Ried

Publications and source records attributed to E Ried.

8 recordsLinked to original sources

Cochleostomy. Long term results.

The original technique of the operation of cochleostomy is described. It should be considered as conservative operation, since it produces an "ostomy" of the cochlear promontory bone, joining the spiral ligament and mucosa of middle ear, without opening of the Cochlea. There is no risk of iatrogenic anacusis. The follow up of 178 cases with more than 10 years after surgery is described. The results are significantly better than endolymphatic sac surgery.

Cochlea↗

Deletion 14q and pericentric inversion 14.

A woman with deletion 14q as well as inversion 14 is presented, and physical signs are compared with those of patients with deletion long arm 13. No previous case of deletion long arm 14 has been published.

Chromosome Deletion↗

[Inverted acoustic reflex in patients with otosclerosis].

The presence of inverted acoustic reflex (IAR) was observed in patients with surgically-confirmed otosclerosis. The characteristics of intensity, stimuli, and thresholds were analyzed. IAR was found in 90% of the patients stimulated on the same side at 110 dB HL with 500, 1000, and 2000-Hz frequencies. The intensity of IAR was higher at 1000-Hz and in patients with mixed hearing loss. We conclude that the detection of IAR in patients with conductive hearing loss and normal tympanic membrane allows otosclerosis to be differentiated from other pathologies that produce ossicular chain fixation. This sign can be used as a diagnostic test because it has a high positive predictive value, 98% in our series.

Adult↗

[Deafness syndrome caused by efferent retrocochlear inhibition].

Under the name of RIED syndrome (retrocochlear inhibition efferent deafness syndrome). I would like to introduce the clinical subject of sudden neurosensorial deafness, or rapidly progressive deafness, which is accompanied by tinnitus, and occasionally by dizziness; all this is related to stressful situations, undergone by tense and perfectionist people who are unable to relax. The possible mechanism of this active efferent inhibition can work whether in one or both ears, from the cortex, going through the medial geniculate body to the low colliculus, and from here to the olivary cochlear set, whose efferent neurons terminate in the external ciliate cells. The possible way in which this efferent retrocochlear inhibition might work, could be in altering the usual operation of the external ciliate cells, which would stop their amplifying and modulating function, causing deafness and tinnitus; this possibly happens because of the immediate alteration of the signal that originates in the internal ciliate cells and that reaches the cortex through the afferent canal; due to the personality of the people that suffer from retrocochlear inhibition efferent deafness syndrome, it is assumed that there could be some disorder in the neurotransmitters; this is very similar to what happens with endogenous depression, and this malfunctioning would take place in the medial geniculate nuclei and inferior colliculus, disturbing the efferent control that this has over the cochlea.

Cochlea↗