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Biomedical subjects

E S RUSSELL

Publications and source records attributed to E S RUSSELL.

At least 19 recordsLinked to original sources
PubMed1964

HEMOGLOBIN SOLUBILITY AND ALPHA-CHAIN STRUCTURE IN CROSSES BETWEEN TWO INBREAD MOUSE STRAINS.

In genetic crosses between mouse strains with low and high solubilities of hemoglobin, solubility segregated as a Mendelian unit and appeared to be determined by the alleles controlling alpha-chain structure. New data eliminate close linkage between the alpha-chain locus and genetic markers in linkage groups Ill, IV, XI, XIII, and XVI. Simplified methods are presented for screening differences inalpha-chain peptides and solubility.

Alleles↗
PubMed1963

ERYTHROID HOMEOSTASIS IN NORMAL AND GENETICALLY ANEMIC MICE: REACTION TO INDUCED POLYCYTHEMIA.

After hypertransfusion, normal ww and anemic WW(T) mice show the same increase in red-cell volume, decrease in reticulocytes, and temporary cessation of blood formation. The time required for the red cell volume to return to the value observed before treatment is the same for both groucps of inice. The cell volume at which new erythrocytes are again released into the circulation depends upon the genotype of functioning blood-forming tissue. Injections of erythropoietin stimulate red cell formation in polycythemic ww mice, but have much less (if any) effect upon polycythemic WW(T) mice.

Anemia↗
PubMed1963

ELECTROPHORETIC PATTERNS OF HEMOGLOBIN FROM FETAL MICE OF DIFFERENT INBRED STRAINS.

Starch gel electrophoretic patterns of hemoglobins from fetal mice of seven inbred strains (two with single, five with diffuse adult hemoglobins), were compared with each other and with electrophoretic patterns of hemoglobins from adults of the same strains. The blood from 15-day-old fetuses of all strains contained four electrophoretically separable heme components. However, there seems to be a difference between strains with single and diffuse adult hemoglobin in the time of emergence of a clear adult pattern.

Animals↗
PubMed1962

New genetically homogeneous background for dystrophic mice and their normal counterparts.

A new type of genetically homogeneous dystrophic and comparable normal mice, from an F(1) hybrid cross between 129/Re-dy and C57BL/6J-dy, is now available as a result of a special breeding program. The clinical manifestations in these F(1) hybrid dystrophics correspond closely with those observed in 129/Re-dydy mice, and they give completely comparable results on a variety of research tests. They are, however, considerably healthier than previously available dystrophics, with a growth rate closer to normal and a greatly increased life-span.

Animals↗