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Biomedical subjects

E Słuszkiewicz

Publications and source records attributed to E Słuszkiewicz.

8 recordsLinked to original sources
PubMed1986

Thyroid function in insulin-dependent diabetic children.

Serum concentrations of thyroxine (T4),3,5,3'-triiodothyronine (T3),3,3',5'-reverse triiodothyronine (rT3),T3-binding capacity (TBC) and TSH were measured in 10 children with diabetes mellitus (mean age: 12.8 +/- 2 yr). The children were without ketoacidosis and serum glucose concentrations were 10.64 +/- 1.68 mmol/l (SD). The mean concentrations of serum T4, T3, rT3, TBC and TSH were 116.35 +/- 10.68 nmol/l (SD), 2.81 +/- 0.33 nmol/l, 0.27 +/- 0.04 nmol/l, 1.087 +/- 0.031 and 3.16 +/- 0.9 mu IU/ml, respectively, which were not significantly different from values of 10 normal control subjects (mean age: 11.6 +/- 2 yr). It seems therefore that the thyroid hormones and TSH levels between well controlled diabetic and healthy subjects of pediatric age do not differ significantly.

Adolescent↗
PubMed1985

Effect of prednison therapy on serum levels of thyroxine (T4), triiodothyronine (T3), reverse triiodothyronine (rT3), T3-binding capacity, basal TSH level and TSH response to thyreoliberin (TRH) in children.

Effects of Prednison therapy on serum levels of T4, T3, rT3, TBC, basal TSH and TSH response to TRH in 10 children (7 with nephrotic syndrome, 3 with trombocytopenia) were studied. It has been found out that corticotherapy (Prednison 1--2 mg/kg/24 h; 2--4 weeks) causes a significant decrease of total T3 concentration together with a considerable rise of reverse T3 and lower T3-binding capacity. Basal TSH level was increased about twice; TSH response to TRH between both groups (without and with Prednison therapy) did not differ significantly. The observations suggest the presence of hypothyroidism during chronic corticotherapy in children.

Child↗
PubMed1980

Suppression of thyroid proliferative response to exogenous TSH by prostaglandin synthesis inhibitors.

The influence of three different inhibitors of prostaglandin (PGs) synthesis (aspirin, indomethacin and hydrocortisone) on the proliferative effect of thyrotropin (TSH) has been investigated. It has been found that all these PGs synthesis inhibitors distinctly inhibit the proliferative response of thyroid follicular cells to TSH. This finding suggests an involvement of PGs in the proliferative action of TSH.

Animals↗