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E Trefelner

Publications and source records attributed to E Trefelner.

3 recordsLinked to original sources

Pneumoparotid due to spirometry.

Pneumoparotid has been described in patients who generate increased intraoral pressures when playing wind instruments, while coughing, and when undergoing dental work. Some patients have intentionally created pneumoparotid to avoid duties at school or in the military, or to gain attention. We describe a patient who developed pneumoparotid during pulmonary function testing. The diagnosis of pneumoparotid depends on a suggestive clinical situation and glandular swelling with or without crepitus. Observation of aerated saliva per Stensen's duct or air in the parotid duct and/or gland by any imaging study is diagnostic if infection with a gas-forming organism can be reasonably excluded. No specific treatment is required, other than the avoidance of predisposing activities.

Adult↗

Meniscal abnormalities in the asymptomatic population at MR imaging.

Two hundred eighty meniscal horns in 64 asymptomatic volunteers in the 2nd to 8th decades of life were analyzed for meniscal abnormalities. Grade 1, 2, and 3 changes were present in essentially all decades. There was at least a 25% prevalence of meniscal signal abnormalities as early as the 2nd decade. The prevalence of meniscal abnormalities increased sharply with age. The prevalence of all signal abnormalities correlated with age, grade 2 changes having the highest correlation coefficient (+.88). The correlation coefficient for grades 1 and 3 changes were +.60 and +.71, respectively. The posterior horn of the medial meniscus had a significantly higher (P less than .02) prevalence of abnormalities than did the other meniscal locations. There was no significant correlation between subject weight or sex and meniscal signal. The authors conclude that there is a baseline prevalence of meniscal signal in the asymptomatic population.

Adolescent↗

Idiopathic growth hormone deficiency: MR findings in 35 patients.

Idiopathic growth hormone deficiency is a disorder that is not clearly understood. We therefore evaluated the MR scans of 35 patients with idiopathic growth hormone deficiency in an attempt to define more clearly the abnormalities of the hypothalamohypophyseal axis, determine the frequency of these abnormalities, and determine whether a relationship exists between the MR findings and the patient's clinical history and endocrine function. Patients with MR abnormalities fell into two groups; those with an ectopic neurohypophysis (15 patients, or 43%), which consisted of a neurohypophysis near the median eminence, absence of the infundibulum, and absence of the normal posterior pituitary bright spot; and those with a small anterior pituitary gland (15 patients, or 43%), which was an isolated finding in five patients and associated with an ectopic neurohypophysis in 10 patients. Examination of endocrine function identified two groups of patients: those with multiple hormone deficiencies and those with isolated growth hormone deficiency. An ectopic neurohypophysis was present in 87% of the first group and 10% of the second group. The anterior pituitary dysfunction in those with an ectopic neurohypophysis was thought to be related to the absent infundibulum, which normally houses the portal system. Our MR findings demonstrated that over 40% of patients with idiopathic growth hormone deficiency have an ectopic neurohypophysis and absence of the infundibulum. We believe that the growth hormone deficiency in these patients is related to the absent infundibulum.

Adolescent↗