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Biomedical subjects

F Depeursinge

Publications and source records attributed to F Depeursinge.

6 recordsLinked to original sources
PubMed1991

[Transesophageal echocardiography. Indications and clinical usefulness].

To optimize the clinical applications of transesophageal echocardiography (TOE), we have compared the contribution of TOE with that of transthoracic echocardiography (TEE). TOE and TTE were performed in 19 outpatients, 17 hospitalized patients and 16 surgical patients (33 males and 19 females, mean age 53.3 +/- 16.8 years). TOE obtained a diagnosis unsuspected from TTE in 12 cases, and additional useful clinical information was obtained by TOE in 15 cases. Surgical treatment was decided on the basis of TOE alone in 8 cases. Patient tolerance of TOE was excellent and no major complications occurred. In conclusion, additional information obtained by TOE was clinically useful in about half of patients (52%), in particular in the diagnosis of cardiogenic embolism and in clinically suspect endocarditis, valvular dysfunction or aortic dissection.

Adult↗
PubMed1988

Respiratory acoustical impedance: a new technique to measure airway response during bronchial inhalation challenges.

Respiratory acoustical impedance, a new method to measure airway obstruction during quiet breathing, was compared with plethysmography and spirometry in 19 asthmatics undergoing a bronchial inhalation challenge with an allergen. Respiratory acoustical impedance and plethysmography were both more sensitive than spirometry in detecting bronchoconstriction. Respiratory acoustical impedance is easily measured during quiet breathing, even in uncooperative subjects; forced expirations, which may alter bronchial tone, are avoided. Respiratory acoustical impedance is sensitive and reliable in measuring acute airway obstruction.

Acoustic Impedance Tests↗
PubMed1985

[Acute myocardial dysfunction in meningococcal septicemia].

A progressive myocardial dysfunction is known to occur in prolonged septic shock. The cases of two patients with acute meningococcal infection and Waterhouse-Friderichsen syndrome are reported. Hemodynamic measurements showed, in the early stage, the classical picture of hyperdynamic septic shock, with increased cardiac output and low systemic vascular resistances. However, rapidly progressive cardiac failure then occurred, with high filling pressures and low cardiac output. One patient went into irreversible shock. In the other the administration of inotropic agents was beneficial, with a slow but progressive improvement in left ventricular function over several weeks. Acute myocarditis has been frequently observed in patients with meningococcal infections. It is suggested that the acute myocardial failure observed in those circumstances is caused by the myocarditis.

Acute Disease↗
PubMed1983

[Measuring bronchial obstruction in intensive care].

The nature and degree of bronchial obstruction is not easy to assess in the intensive care unit. The classic airway resistance measurement technique cannot be performed without heavy equipment and the patient's cooperation. On the basis of the forced oscillations technique, measurement of acoustical respiratory impedance is an extension, in a higher frequency range, of earlier dynamic compliance measurements, which are known to be sensitive to peripheral airway obstruction but unsuitable for critically ill patients. The main advantage of respiratory impedance measurement is simplicity, since the patient simply breathes freely for a minute in the apparatus. This method contributes an effective means of quantifying evolution and treatment during an asthmatic attack. It can also be used to monitor left heart failure.

Acute Disease↗
PubMed1981

[Myocardial dysfunction in septic shock].

Septic shock is known to induce progressive myocardial dysfunction characterized hemodynamically by a decrease in cardiac output and a rise in ventricular filling pressures. Both ventricles are affected. Benefit from inotropic agents is inconsistent. Identification of the factors responsible for the progressive cardiac deterioration is rendered difficult by the complexity of clinical situations. Experimental models demonstrate a progressive inhibition of contractility. Several arguments suggest the possible role of interstitial myocardial edema in altering the microcirculation, decreasing substrate delivery and diminishing ventricular compliance. The role of a myocardial depressant factor has still to be demonstrated.

Adult↗