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Biomedical subjects

F Jaisle

Publications and source records attributed to F Jaisle.

At least 19 recordsLinked to original sources
PubMed1981

[The operative treatment of enterocele and prolapse of the vaginal vault (author's transl)].

Between 1969 and 1980 11 operations according to Williams-Richardson, 23 abdominal sacropexies according to Wagner-Kuestner and 4 operations according to Amreich-II were performed for the treatment of enteroceles and prolapse of the vaginal vault. The combination of these operative methods with colpoperineorraphies and Marshall-Marchetti-Krantz operations and lyodura ribbon operations according to Zoedler for the urethro-vesical angle is described. Continence an elevation of the vaginal fornix was obtained by the operation according to Williams-Richardson and by the fixation of the vagina to the sacrospinal ligament. With the Williams-Richardson operation 1 enterocele occurred which was corrected with the vaginal fixation to the sacro-spinal ligament. Following fixation to the promontary 2 enteroceles, 4 cystocele, and one rectocele occurred in 4 patients. Three of these had urinary incontinence. The complications are described. The vaginal fixation to the sacro-spinal ligament according to Amreich II at times combined with the lyodura sling operation of Zoedler is today the preferred operative method.

Female↗
PubMed1981

[The prevention and treatment of recurrent urinary stress incontinence with the suspension according to Zoedler (author's transl)].

115 patients with severe urinary stress incontinence were operated according to the method of Zoedler with a nylon ribbon (31 cases) and a lyodura ribbon (84 cases). There were 56 primary operations and 59 operations for recurrence. In 13 patients the operation was combined with a fixation to the promentory according to Kuestner-Wagner. Continence was obtained in 90.3% of the cases at the end of one year in 84.2% of the cases at the end of three years and in 83% of the cases at the end of five years. The recurrence rate for the operations for recurrent stress incontinence was 20.6% the recurrence rate in the primary operations was 9.1%. The complications were described. The lyodura sling procedure is today primarily practiced as an operation for recurrent stress incontinence.

Aged↗
PubMed1980

[Excessive hyperlipidemia during pregnancy (author's transl)].

1) In two patient with excessive hyperlipidemia diet in the post-partum days resulted in a rapid reduction of the high neutral fats in the serum which were 6,150 mg/dl and 6,290 mg/dl respectively. In one patient the hyperlipidemia was reduced by diet during the pregnancy. The course of the pregnancies and the puerperium were uneventful. Complications during delivery were not related to the hyperlipidemia. Coagulation defects were not tested.--2) Excessively high serum lipid values of the mother only had a minor influence on the serum lipid values of the mother only had a minor influence on the serum lipid values of the infants. The infants had slightly elevated values compared to controls. One infant was tested at age 7 months and showed definitely elevated lipids with higher values than neonatally. A familial hyperlipidemia was confirmed in this case.--3) The milk fat values (Total Cholesterol and Neutral fats) were not influenced by the hyperlipidemia. Breast feeding was therefore safe.--4) Both infants of the mothers with hyperlipidemia had neonatal hyperbilirubinaemia with values of maximal 16.2 mg% and 16.7 mg% which required phototherapy.

Adult↗
PubMedDate not supplied

[Etiology of cerebral palsy].

The "perinatal asphyxia" is regarded to be one of the causes of cerebral palsy, though in the very most of the children with cerebral palsy there is found no hypoxia during labour. It should be mentioned, that the definition of "perinatal" and "asphyxia" neither are unic nor concret. And also there is no correlation between nonreassuring fetal heart rate patterns and acidosis in fetal blood with the incidence of cerebral palsy. Numerous studies in pregnant animals failed in proving an acute intrapartal hypoxia to be the origin of the cerebral palsy. Myers (1975) describes four patterns of anatomic brain damage after different injuries. Only his so called oligo-acidotic hypoxia, which is protracted and lasts over a longer time is leading to brain injury, which can be regarded in analogy to the injury of children with cerebral palsy. Summarising the update publications about the causes of cerebral palsy and the studies in pregnant animals there is no evidence that hypoxia during labour may be the cause of cerebral palsy. There is a great probability of a pre(and post-)natal origin of brain injury (for instance a periventricular leucomalacia found after birth) which leads to cerebral palsy. Short after labour signs of a so called "asphyxia" may occur in addition to this preexisting injury and misrepresent the cause of cerebral palsy. Finally the prepartal injury may cause both: Cerebral palsy and hypoxia.

Acidosis↗